Tuesday, August 16, 2022
  Béchamp or Pasteur?  

Book 1 ♦ Pasteur: Plagiarist, Impostor – The Germ Theory Exploded

Introduction
A Note from the Publisher

This volume contains new editions of two books which have been available only sporadically in the decades since their publication. R. Pearson’s Pasteur: Plagiarist, Imposter was originally published in 1942, and is a succinct introduction to both Louis Pasteur and Antoine Béchamp, and the reasons behind the troubled relationship that they shared for their entire working lives.

Whereas Pearson’s work is a valuable introduction to an often complex topic, it is Ethel Douglas Hume’s expansive and well-documented Béchamp or Pasteur? A Lost Chapter in the History of Biology which provides the main body of evidence. It covers the main points of contention between Béchamp and Pasteur in depth sufficient to satisfy any degree of scientific or historical scrutiny, and it contains, wherever possible, detailed references to the source material and supporting evidence. Virtually no claim in Ms Hume’s book is undocumented – to have access to more material, one would need to be able to read French, and go to the original source material.

The reader will soon discern that neither Mr Pearson nor Ms Hume could ever be called fans of Pasteur or his ‘science’. They both declare their intentions openly; that they wish to contribute to the undoing of a massive medical and scientific fraud. The publication of this present edition of their work is undertaken with complete empathy for that intention.

The text of both books has been comprehensively re-edited – for style more than content – the intention being to make for easier reading than the style of language used in the first half of the twentieth century would otherwise allow. I hope that the end result is an improvement, and that the authors would approve. I think they would.

I intend to publish in due course a collection of modern writings on Béchamp and pleomorphism. Any researchers who wish to be involved in such a project by contributing material or otherwise can make contact via the web site at www.bechamp.org.

David Major
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November 2006


This edition copyright ©2006 Bechamp.org

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Author’s Preface

It is a serious matter to attack the reputation of a famous man, especially one who has posed – and been accepted – as one of the world’s greatest scientists. For many years, Pasteur has been looked upon as a founder and leader in serology; but it is always pertinent to look into the beginnings of any subject on which there is a difference of opinion, with the hope of finding the truth in the matter.

The writer has made an effort in his prior books and pamphlets to show that the germ theory is false, and that illness is practically always due to errors of diet or manner of living, the germs being present solely as scavengers of dead and waste tissues and foods, and not as the cause of the disease.

However, the erroneous belief that germs cause disease and must be controlled or eliminated before it can be cured is so widespread as to close the minds of many people to any other ideas on this subject. For this reason it seems that a thorough investigation of this idea, the grounds on which it is based – and even the bona fides of those who started it on its way – is necessary before any sane ideas as to the proper treatment of disease can be widely promulgated.

When Ethel Douglas Hume’s Béchamp or Pasteur? appeared in 1923, it seemed to be just the thing that would fill this gap and end the use of serums and other biologicals forever. But it is now 19 years since that book, which should have marked an epoch in the healing arts, was published. It did not receive the attention it deserved in medical circles and, though it is now in its second edition,1 the medical profession are pushing biologicals harder than ever.

Hence it seems appropriate to go over the subject in order to show the truth regarding the falsity of Pasteur’s ideas and claims to fame, and the fraudulent basis on which the germ theory rests, as was so well shown by Ms Hume in Béchamp or Pasteur?, and to add other facts and statistics that support the idea that the germ theory is false, in the hopes that it may receive wider circulation and more general attention, and possibly lead to a complete overhauling of the question of the treatment of disease, especially regarding serology.

The translations from the French, and other material in Chapters 2, 3, 4 and 5 not otherwise credited, are from Béchamp or Pasteur? by Ethel Douglas Hume.

In closing, I wish to acknowledge my indebtedness to the Reverend and Mrs Wilber Atchison of Chicago for many suggestions and valuable assistance in the preparation of the manuscript. Miss L. Loat, secretary of the National Anti-Vaccination League of London, has also been very kind, responding to every request for information with more than could be used, some of it being especially compiled at the cost of considerable effort.

R. B. Pearson
January 15th, 1942


1. Published by the National Anti-Vaccination League, London, England.

Contents

Chapter 1
Chapter 2
Chapter 3
Chapter 4
Chapter 5
Chapter 6
Chapter 7
Chapter 8
Chapter 9
Chapter 10
The Prior History of the Germ Theory
Béchamp, Pasteur, and Fermentation
Vinous Fermentation
Béchamp’s Microzymas or ‘little bodies'
Silkworm Disease: Another Steal!
Pasteur also a Faker: Antisepsis
Are Biologicals Injurious?
Animal Serology: Anthrax
Statistics Real Immunity
Real Immunity
  3
5
14
15
21
27
33
45
55
66
Photos

10-1
Antoine Béchamp with his wife Clementine and son Joseph.
  10-4
Professor Béchamp (r) and an unknown colleague
 
10 5
Louis Pasteur
10 6
Ethel Douglas Hume
Title Page

Pasteur: Plagiarist, Impostor

The Germ Theory Exploded

R.B. PEARSON

First published in 1942

Chapter 1 ♦ The Prior History of the Germ Theory

If you explore the history of the medical profession and the various ideas regarding the cause of disease that were held by leading physicians before Pasteur first promulgated his notorious ‘germ theory’, you will find convincing evidence that Pasteur discovered nothing, and that he deliberately appropriated, falsified and perverted another man’s work.

The ‘germ theory’, so-called, long antedated Pasteur – so long, in fact, that he was able to present it as new – and he got away with it!

F. Harrison, Principal Professor of Bacteriology at Macdonald College (Faculty of Agriculture, McGill University), Quebec, Canada, wrote an Historical Review of Microbiology, published in Microbiology, a text book, in which he says in part:

“Geronimo Fracastorio (an Italian poet and physician, 1483 – 1553) of Verona, published a work (De Contagionibus et Contagiosis Morbis, et eorum Curatione) in Venice in 1546 which contained the first statement of the true nature of contagion, infection, or disease organisms, and of the modes of transmission of infectious disease. He divided diseases into those which infect by either immediate contact, through intermediate agents, or at a distance through the air. Organisms which cause disease, called seminaria contagionum, he supposed to be of the nature of viscous or glutinous matter, similar to the colloidal states of substances described by modern physical chemists. These particles, too small to be seen, were capable of reproduction in appropriate media, and became pathogenic through the action of animal heat. Thus Fracastorio, in the middle of the sixteenth century, gave us an outline of morbid processes in terms of microbiology.”

For a book published more than three hundred years before Pasteur ‘discovered’ the germ theory, this seems to be a most astonishing anticipation of Pasteur’s ideas, except that – not having a microscope – Fracastorio apparently did not realize that these substances might be individual living organisms.

According to Harrison, the first compound microscope was made by H. Jansen in 1590 in Holland, but it was not until about 1683 that anything was built of sufficient power to show up bacteria. He continues:

“In the year 1683, Antonius van Leenwenhoek, a Dutch naturalist and a maker of lenses, communicated to the English Royal Society the results of observations which he had made with a simple microscope of his own construction, magnifying from 100 to 150 times. He found in water, saliva, dental tartar, etc., entities he named animalcula. He described what he saw, and in his drawings showed both rod-like and spiral forms, both of which he said had motility. In all probability, the two species he saw were those now recognized as bacillus buccalis maximus and spirillum sputigenum.

“Leenwenhoek’s observations were purely objective and in striking contrast with the speculative views of M. A. Plenciz, a Viennese physician, who in 1762 published a germ theory of infectious diseases. Plenciz maintained that there was a special organism by which each infectious disease was produced, that micro-organisms were capable of reproduction outside of the body, and that they might be conveyed from place to place by the air.”

Here is Pasteur’s great thought in toto – his complete germ theory – and yet put in print over a century before Pasteur ‘thought of it’, and published it as his own!

Note how concisely it anticipates all Pasteur’s ideas on germs. While there seems to be no proof that Plenciz had a microscope, or knew of Leenwenhoek’s animalcula, both are possible, indeed likely, as he was quite prominent; and he, rather than Pasteur, should have any credit that might come from such a discovery – if the germ theory has any value. This idea, which, to the people of that time at least, must have accounted easily and completely for such strange occurrences as contagion, infection and epidemics, would have been widely discussed in the medical or scientific circles of that time, and in literature available to Pasteur.

That it was widely known is indicated by the fact that the world-famous English nurse, Florence Nightingale, published an attack on the idea in 1860, over 17 years before Pasteur adopted it and claimed it as his own.

She said of ‘infection’1:

“Diseases are not individuals arranged in classes, like cats and dogs, but conditions, growing out of one another.

“Is it not living in a continual mistake to look upon diseases as we do now, as separate entities, which must exist, like cats and dogs, instead of looking upon them as conditions, like a dirty and a clean condition, and just as much under our control; or rather as the reactions of kindly nature, against the conditions in which we have placed ourselves?

“I was brought up to believe that smallpox, for instance, was a thing of which there was once a first specimen in the world, which went on propagating itself, in a perpetual chain of descent, just as there was a first dog, (or a first pair of dogs) and that smallpox would not begin itself, any more than a new dog would begin without there having been a parent dog.

“Since then I have seen with my own eyes and smelled with my own nose smallpox growing up in first specimens, either in closed rooms or in overcrowded wards, where it could not by any possibility have been ‘caught’, but must have begun.

“I have seen diseases begin, grow up, and turn into one another. Now, dogs do not turn into cats.

“I have seen, for instance, with a little overcrowding, continued fever grow up; and with a little more, typhoid fever; and with a little more, typhus, and all in the same ward or hut.

“Would it not be far better, truer, and more practical, if we looked upon disease in this light (for diseases, as all experience shows, are adjectives, not noun-substantives):

  • “True nursing ignores infection, except to prevent it. Cleanliness and fresh air from open windows, with unremitting attention to the patient, are the only defence a true nurse either asks or needs.
  • “Wise and humane management of the patient is the best safeguard against infection. The greater part of nursing consists of preserving cleanliness.
  • “The specific disease doctrine is the grand refuge of weak, uncultured, unstable minds, such as now rule in the medical profession. There are no specific diseases; there are specific disease conditions.”

Here you have Florence Nightingale, the most famous nurse in history, after life-long experience with infection, contagion and epidemics, challenging the germ theory 17 years before Pasteur put it forward as his own discovery! (See Chapter 8, p.50).

She clearly understood it and its utter fallacy better before 1860 than Pasteur did, either in 1878 or later!

Now, to see what a parasite Pasteur was on men who did things, let us digress and go back a few years, to the time when the study of germs was an outgrowth of the study of fermentation.


1. Notes on Nursing, 1st ed., 1860, p.32

Chapter 2 ♦ Béchamp, Pasteur, and Fermentation1

About 1854, Professor Pierre Jacques Antoine Béchamp, one of France’s greatest scientists, then Professor at the School of Pharmacy in the Faculty of Science at Strasbourg, later (1857-75) Professor of Medical Chemistry and Pharmacy at the University of Montpelier, a member of many scientific societies, and a Chevalier of the Legion of Honor, took up the study of fermentation.

He had succeeded in 1852 in so reducing the cost of producing aniline as to make it a commercial success, and his formula became the basis of the German dye industry. This brought him some fame – and many more problems to solve.

Up to this time, the idea prevailed that cane sugar, when dissolved in water, was spontaneously transformed at an ordinary temperature into invert sugar, which is a mixture of equal parts of glucose and fructose, but an experiment with starch had caused him to doubt the truth of this idea.

Therefore in May, 1854, Béchamp undertook a series of observations on this change, which came to be referred to as his ‘Beacon Experiment’. In this experiment, he dissolved perfectly pure cane sugar in water in a glass bottle containing air, but tightly stoppered. Several other bottles contained the same solution, but with a chemical added.

In the solution without any added chemical, moulds appeared in about thirty days, and inversion of the sugar in this bottle then went on rapidly, but moulds and inversion did not occur in the other bottles containing added chemicals. He measured the inversion frequently with a polariscope.

These observations were concluded on February 3, 1855, and his paper was published in the Report of the French Academy of Science for the session of February 19, 1855.2

This left the moulds without an explanation, so he started a second series of observations on June 25, 1856 (at Strasbourg) in order to determine their origin, and on March 27, 1857, he started a third series of flasks to study the effects of creosote on the changes. Both series were ended at Montpelier on December 5, 1857.

In the second series he spilled a little liquid from flasks 1 and 2 during manipulation, so these two flasks contained a little air in contact with the liquid. In these two flasks, moulds soon appeared, and alteration in the medium ensued.

He also found that the changes were more rapid in the flask in which the mould grew more rapidly.

In the other nine flasks there was no air, no mould formed, and no inversion of the sugar occurred; plainly air was needed for the moulds and inversion to occur. This proved beyond any possibility of doubt that the moulds and inversion of the sugar could not be ‘spontaneous’ action, but must be due to something in the air admitted to the first two flasks.

Yet Pasteur later called fermentation

“life without air, or life without oxygen.”3

At this time, it was quite generally believed that fermentation could not take place except in the presence of albuminoids, which were in general use by Pasteur and others as part of their solutions. Hence, their solutions could have contained these living organizations to start with.

Béchamp’s solutions contained only pure cane sugar and water, and when heated with fresh-slaked lime did not disengage ammonia – ample proof that they contained no albumen. Yet moulds, obviously living organisms, and therefore containing albuminoid matter, had appeared in these two solutions.

Béchamp proved to his own satisfaction that these moulds were living organisms and that cane sugar was inverted, as he said

“… only in proportion to the development of moulds … these elementary vegetations then acting as ferments.”4

Pasteur, apparently overlooking the air contact, challenged Béchamp’s statements, saying:

“… to be logical, Béchamp should say that he has proved that moulds arise in pure sugared water, without nitrogen, phosphates or other mineral elements, for that is an enormity that can be deduced from his work, in which there is not the expression of the least astonishment that moulds have been able to grow in pure water with pure sugar without any other mineral or organic principles.”5

Béchamp’s retort to this was:

“A chemist au courant with science ought not to be surprised that moulds are developed in sweetened water, contained in contact with air in glass flasks. It is the astonishment of Pasteur that is astonishing”6

As Béchamp started with no nitrogen whatever except what was in the air in the first two flasks, it is probably the first time any growth or any kind of organism was proved to have absorbed nitrogen from the air. Apparently Pasteur could not grasp this idea!

In the preface to his last book, The Blood and its Third Anatomical Element, Béchamp says that these facts impressed him in the same way that the swing of the cathedral lamp had impressed Galileo. He realized that some living organisms had been carried into these two flasks in the small amount of air admitted, and acting as ferments had produced the mould and the inversion in the sugar. He compared the transformation of cane sugar in the presence of moulds to that produced upon starch by diastase, the ferment that converts starch into sugar.

He sent his report on these findings to the Academy of Science in December 1857, and an extract was published in its reports of January 4, 1858, though the full paper was not published until September of that year.7

He says of these experiments:

“By its title the memoir was a work of pure chemistry, which had at first no other object than to determine whether or not pure cold water could invert cane sugar and if, further, the salts had any influence on the inversion. But soon the question, as I had foreseen, became complicated; it became at once physiological and dependent upon the phenomena of fermentation and the question of spontaneous generation. Thus from the study of a simple chemical fact, I was led to investigate the causes of fermentation, and the nature and origin of ferments.”8

Although Schwann had suggested airborne germs in about 1837, he had not proved his ideas; now Béchamp proved them to exist.

Yet Pasteur in his 1857 memoirs still clings to the idea that both the moulds and ferments ‘take birth spontaneously’, although his solutions all contained dead yeast or yeast broth which might have carried germs or ferments from the start.

He does conclude that the ferment is a living being, yet states that this ‘cannot be irrefutably demonstrated’.9

But Béchamp had demonstrated it ‘irrefutably’ in his paper, and had also proved that water alone caused no alteration, there was no spontaneous alteration, and that moulds do not develop, nor inversion occur, without contact with the air; thus some airborne organism must cause the moulds and the inversion.

According to Miss Hume, Béchamp was also the first to distinguish between the ‘organized’ or living ferment and the soluble ferment which he obtained by crushing the moulds, and which he found to act directly on the sugar, causing rapid inversion.

He named this substance zymase, in a paper Memoirs on Fermentation by Organized Ferments, which he read before the Academy of Science on April 4, 1864.10

Strange to say, exactly the same word is used by others whom various encyclopaedias have credited with this discovery in 1897, over 30 years later!

In this paper he also gave a complete explanation of the phenomena of fermentation as being due to the nutrition of living organisms; i.e. a process of absorption, assimilation, and excretion.

In the preface to The Blood and its Third Anatomical Element, Béchamp says:

“It resulted that the soluble ferment was allied to the insoluble by the relation of product to producer; the soluble ferment being unable to exist without the organized ferment, which is necessarily insoluble.

“Further, as the soluble ferment and the albuminoid matter, being nitrogenous, could only be formed by obtaining the nitrogen from the limited volume of air left in the flasks, it was at the same time demonstrated that the free nitrogen of the air could help directly in the synthesis of the nitrogenous substance of plants – which up to that time had been a disputed question.

“Thus it became evident that since the material forming the structure of moulds and yeast was elaborated within the organism, it must also be true that the soluble ferments and products of fermentation are also secreted there, as was the case with the soluble ferment that inverted the cane sugar. Hence I became assured that that which is called fermentation is in reality the phenomena of nutrition, assimilation and disassimilation, and the excretion of the products disassimilated.”

He explained further:

“In these solutions there existed no albuminoid substance; they were made with pure cane sugar, which, heated with fresh-slaked lime, does not give off ammonia. It thus appears evident that airborne germs found the sugared solution to be a favourable medium for their development, and it must be admitted that the ferment is here produced by the generation of fungi.

“The matter that develops in the sugared water sometimes presents itself in the form of little isolated bodies, and sometimes in the form of voluminous colourless membranes which come out in one mass from the flasks. These membranes, heated with caustic potash, give off ammonia in abundance.”

This proved that albuminoids were present, hence the little bodies were living matter. It also proves that Professor Béchamp understood the formation and growth of moulds and ferments in 1857, years before Pasteur comprehended these physiological processes!

In 1859, over a year after Béchamp’s paper covering his 1857 experiments was printed, Pasteur started another experiment more in line with Béchamp’s ideas; in fact it was apparently inspired by them.

He omitted all yeast but used ammonia, which contains nitrogen, in his solutions, and then ascribed the origin of lactic yeast to the atmospheric air. He was surprised that animal and vegetable matter should appear and grow in such an environment.

He says:

“As to the origin of the lactic yeast in these experiments, it is solely due to the atmospheric air; we fall back here upon facts of spontaneous generation.”

After asserting that excluding atmospheric air or boiling the solution will prevent the formation of organisms, or fermentations, he says:

“On this point, the question of spontaneous generation has made progress.”

In a still later memoir11 plainly inspired by Béchamp’s Beacon Experiment, Pasteur again constantly refers to the spontaneous production of yeasts and fermentation.

There is no question but that he still believed in spontaneous generation of germs and ferments at this time, and his reasoning appears somewhat childish when compared to Béchamp’s work.

However, in 1860, he started another experiment in which he prepared 73 phials of unfermented liquid to expose at various points on a much advertised-in-advance trip. He opened and resealed various phials at different places, the last twenty on the Mer de Glace above Chamonix.

He practically repeated Béchamp’s experiments here, but of course he had to use a different and more spectacular method to get attention.

From this time on, he veered away from spontaneous generation, and began to explain the same occurrences (fermentation) as being caused by germs in the air.

Paul de Kruif in Microbe Hunters (a grandiose attempt to exalt some of the original experimenters in serumology), glosses over Pasteur’s willingness to steal credit for the ideas of others, and after describing his use, without credit, of Ballard’s suggestion of the swan neck bottle to admit dust-free and germ-free air into a flask, says of this ‘high Alps’ experiment:

“Then Pasteur invented an experiment that was – so far as one can tell from a careful search through the records – really his own. It was a grand experiment, a semi-public experiment, an experiment that meant rushing across France in trains, it was a test in which he had to slither on glaciers.” (p.83)

However, de Kruif doubted thoroughly that it was Pasteur’s, and well he might! Yet little did he realize how few of Pasteur’s foolhard claims were either his own or, in fact, even true in any particular.

In a discussion of spontaneous generation at the Sorbonne during a meeting on November 22, 1861, Pasteur had the nerve to claim, in the presence of Professor Béchamp, all credit for the proof that living organisms appeared in a medium devoid of albuminoid matter! Béchamp asked him to admit knowledge of Béchamp’s 1857 work, but did not charge him with plagiarism, and Pasteur evaded the question, merely admitting that Béchamp’s work was ‘rigidly exact’. This was not an accident, but deliberate, premeditated fraud; however, Béchamp was too much of a gentleman to make any unpleasant charges.

That it took several more years to get the spontaneous generation idea entirely out of Pasteur’s head is indicated by the article on Pasteur in the 14th Edition of the Encyclopaedia Britannica, which says:

“The recognition of the fact that both lactic and alcohol fermentation were hastened by exposure to air naturally led Pasteur to wonder whether his invisible organisms were always present in the atmosphere or whether they were spontaneously generated. By a series of intricate experiments, including the filtration of air and the famous exposure of unfermented liquids to the pure air of the high Alps, he was able to declare with certainty in 1864 that the minute organisms causing fermentation were not spontaneously generated but came from similar organisms with which ordinary air was impregnated.”12

Here it is again – not until 1864 did he give up his idea of spontaneous generation – and the high Alps adventure was only high theatre, well advertised in advance, to enable him to grab Béchamp’s discovery, and yet have some ‘new stuff’ to attract attention to himself. Of course, he could not follow exactly the same methods; some one might bring up Béchamp’s memoirs, hence the ‘high Alps’ and ‘slithering on glaciers’.

His experiments made in 1859 also indicated knowledge of Béchamp’s work without albuminoids, and his evasion of Béchamp’s question at the Sorbonne meeting in 1861 lends further support to such a belief, while his attacks on Béchamp suggest that he recognized a rival and was keenly jealous.

Note that this final acceptance of ideas that Béchamp had brought forward six years earlier did not come until after Béchamp had published his complete paper, with a full and most thoroughly proven explanation of the processes of fermentation.

However, Pasteur had, on completion of his ‘high Alps’ experiment in 1860, accepted, or had begun to accept, the idea that germs of the air caused fermentation; and soon he leaped way ahead to the conclusion that these germs also caused disease, as Plenciz had suggested about a hundred years before!

Of this idea, he had no more proof than Plenciz, except that it was now known there were germs in existence, which Plenciz, apparently, did not prove.

Although Béchamp had made clear the physiological nature of fermentation in his paper on his 1857 experiments (published in 1858), and had given more complete details in his 1864 paper, Pasteur apparently had not fully grasped its true nature as late as 1872, when he published a paper in which he stated:

“That which separates the chemical phenomenon of fermentation from other acts and especially from the acts of ordinary life is the decomposition of a weight of fermentative matter much greater than the weight of the ferment.”13

Could anyone make such a statement who really understood the true nature of fermentative action? Apparently Pasteur did not!

In collaboration with Professor Estor, Béchamp answered this with an effort to make the nature of fermentation clear, in a paper printed on p.1523 of the same volume of the Comptes Rendus, in which he said:

“Suppose an adult man to have lived a century, and to weigh on average 60 kilograms. He will have consumed over that time, besides other foods, the equivalent of 20,000 kilograms of flesh, and produced about 800 kilograms of urea.

“Of course, there is no suggestion that this mass of flesh and urea could at any moment of his life form part of his being. Just as a man consumes all that food only by repeating the same act a great many times, the yeast cell consumes the great mass of sugar only by constantly assimilating and disassimilating it, bit by bit. Now, that which only one man will consume in a century, a sufficient number of men would absorb in a day.

“It is the same with the yeast; the sugar that a small number of cells would consume in a year, a greater number would destroy in a day. In both cases, the more numerous the individuals, the more rapid the consumption.”14

Is that not clear enough, even for a man whose diploma was marked ‘mediocre in Chemistry’ (i.e. Pasteur) to comprehend? It seems that a child should be able to understand it.

Yet Pasteur repeated his statement four years later in Etudes sur la Bier (1876), so Béchamp’s clear explanation apparently failed to have any effect – at least on him.

Here is proof that from eight to fourteen years after Béchamp had completely disclosed the physiological nature of fermentation and described its action in minute detail, Pasteur had not yet grasped the facts regarding the process!

In its article on fermentation, the Encyclopaedia Britannica says:

“Fermentation, according to Pasteur, was caused by the growth and multiplication of unicellular organisms out of contact with free oxygen, under which circumstances they acquire the power of taking oxygen from chemical compounds in the medium in which they are growing. In other words, ‘fermentation is life without air, or life without oxygen’. This theory of fermentation was materially modified in 1892 and 1894 by A. J. Brown, who described experiments which were in disagreement with Pasteur’s dictum.”15

As did Béchamp over 35 years earlier – in 1855 and 1858 – and Pasteur appropriated and perverted his ideas.

Pasteur also jumped to the conclusion that each kind of fermentation had one specific germ, while Béchamp proved that each micro-organism might vary its fermentative effect in conformity with the medium in which it finds itself. He also showed that these micro-organisms, under varying conditions, might even change their shape, as has been recently proved so conclusively by F. Loehnis and N. R. Smith of the U.S. Dept. of Agriculture and others.16

Pasteur, however, proceeded to classify his germs and label each with a definite and unalterable function, wherein he was wrong again, as we shall soon see.

1. All quotations in Chapters 2, 3, 4, and 5, unless otherwise credited, are taken from Béchamp or Pasteur? by Ethel Hume, originally published by the National Anti-Vaccination League, London, England, and included in this book.
     Miss Hume conducted a thorough investigation of the writings of both Béchamp and Pasteur, and makes some astounding disclosures of plagiarism on the part of the latter, many of which are quoted herein.
     Also, the translations from the French used in these four chapters are all taken from Béchamp or Pasteur?

2. Comptes Rendus de l’Academie des Sciences, 40, p.436

3. Encyclopaedia Brittanica, 11th ed. 10, p.275

4. Comptus Rendus de l’Academie des Sciences, 46, p.44

5. Etudes sur la Biere, 1876, p.310

6. Les Microzymas, p.87

7. Annales de Chimie et du Physique, 3rd series, 54, p.28

8. Les Microzymas, p.55

9. Comptes Rendus, 45, p.1032;
     see also Annales de Chimie et du Physique, 3rd series, 52, p.404

10. Comptes Rendus, 58, p.601

11. Annales de Chimie et du Physique, April 1860

12. Encyclopaedia Britannica, 14th ed., 17, p.357

13. Comptes Rendus, 75, p.785 (session of Sept. 30th, 1872)

14. ibid., p.1523 (session of Dec. 2nd, 1872)

15. Encyclopaedia Britannica, 11th ed., 10, p.275

16. Journal of Agricultural Research, July 31, 1916, p.675.

Chapter 3 ♦ Vinous Fermentation

Another step that went along with the work on fermentation in general was the discovery of the causes of diseases in French grapes. Béchamp, hearing of the commotion over this trouble in the vineyards, quietly took up a study of it in 1862, the year before Pasteur turned his attention to the subject.

Béchamp exposed to contact with air:

  1. grape-must as found on the vines;
  2. grape-must filtered; and
  3. grape-must decolorized by animal charcoal.

They all fermented, but not equally so, and the moulds or ferments developed were not identical in these three experiments, which of course caused him to seek a reason for this.

On further experiments, with the rigid exclusion of all air (the whole healthy grapes, with stalks attached, being introduced directly from the vine into boiled sweetened water, cooled with carbonic acid gas bubbling through it), fermentation took place, and was completed in this medium, proving that air was not required. Hence the ferment must have been carried on the grapes, and was not airborne.

Professor Béchamp concluded that the organism causing the must to ferment must be carried on the grape, its leaves, or the vines, and that it might also be an organism injurious to the plants.

He published a volume on vinous fermentation in 1863, entitled Lecons sur la Fermentation Vineuse et sur la Fabrication du Vin, in which he discussed the subject.

He also presented two papers on the making of wine to the Academy, entitled Sur les Acids du Vin and Sur l’utilité et les Inconvienient du Cuvages Prolongés dans la Fabrication du Vin – Sur la Fermentation Alcoolique dans cette Fabrication.1

In October 1864 he presented a communication to the Academy of Science on The Origin of Vinous Fermentation, an exhaustive account of the experiments described above.2

This paper was a complete study of the subject, in which he proved that vinous fermentation was due to organisms found on the skins of grapes and also often found on the leaves and other parts of the vine. Hence at times, diseased vines might affect the quality of the fermentation and the resulting wine.

So by October 1864, Béchamp had several papers in print, but what was Pasteur up to?

In 1862 Pasteur was admitted to the French Academy through the influence of Biot and the Mineralogical Section, which based its nomination and support on Pasteur’s past work on crystallography; yet many attacks were made on his treatment of that subject, and he soon took the advice of friends to drop this line of work.

In March 1863, he met the Emperor and was soon sent to the vineyards to study the grape disease, with the prestige of having the Emperor’s backing.

He published several papers on the vines and their troubles in the latter part of 1863 and in 1864, but apparently was still promoting his spontaneous generation theory which Béchamp had so completely exploded in 1858, and he did not guess correctly as to the cause of the trouble with the vines.

In 1865 he offered five papers, and others came later, but he does not seem to have hit on the right answer to the problem until 1872, when he made the great discovery that Béchamp was right again! In this year, Pasteur presented a memoir entitled New Experiments to Demonstrate that the Yeast Germ that Makes Wine comes from the Exterior of Grapes.3

As Béchamp had made the same statement in his 1864 paper and it had not been disproven in the intervening eight years, it was a pretty safe bet for Pasteur to make!


1. Comptus Rendus, 57, pp.496, 674

2. ibid, 59, p.626 (session of Oct. 10th, 1864)

3. ibid, 74, p.781 (session of Oct. 7th, 1872)

Chapter 4 ♦ Béchamp’s Microzymas or ‘little bodies'

As shown in the second chapter, Béchamp was the first to prove that the moulds accompanying fermentation were, or contained, living organisms, and could not be spontaneously generated, but must be an outgrowth of some living organism carried in the air.

This much was in his 1858 memoir, six years before Pasteur came to the same conclusions.

Being the first to realize that these moulds or ferments were living organisms, he naturally was also the first to attempt to determine their true nature and functions, and their origins.

On putting some under the microscope, he noted a diversity in appearance of the moulds and was soon involved in a study of cell life.

In his earlier experiments, Béchamp had used several salts, including potassium carbonate, in the presence of which the inversion of cane sugar did not take place. But when he repeated this experiment using calcium carbonate (common chalk) instead of the potassium carbonate, he found that inversion of the cane sugar did take place, even when creosote was added. This observation was so unexpected that he omitted it from his earlier memoir in order to verify it before publication of the fact.

In carefully controlled experiments he found that when chemically pure calcium carbonate, CaCO3, was added to his sugar solutions, no inversion took place, but when ordinary chalk, even that chipped from the native rock without access of air, was used, inversion always occurred.

On heating the common chalk to 300 degrees, he found that it lost its powers of fermentation, and on examining more of the unheated common chalk under the microscope, he found it contained some ‘little bodies’ similar to those found in prior observations, and which he found did not exist in the chemically pure CaCO3, nor in the chalk that had been heated.

These ‘little bodies’ had the power of movement and were smaller than any of the microphytes seen in fermentation or moulds, but were more powerful ferments than any he had encountered previously.

Their power of movement and production of fermentation caused him to regard them as living organisms.

He advised Dumas of his discovery of living organisms in chalk in December 1864, and later, on September 26, 1865, he wrote a letter which Dumas published. He stated:

“Chalk and milk contain already developed living beings, which is proved by the fact that creosote, employed in a non-coagulating dose, does not prevent milk from finally turning, nor chalk, without extraneous help, from converting both sugar and starch into alcohol and then into acetic acid, tartaric acid, and butyric acid…”1

Which was ample proof that there was a ferment, a living organism, present in both milk and chalk. He said of these:

“The naturalist will not be able to distinguish them by a description; but the chemist and also the physiologist will characterize them by their function.”2

Professor Béchamp found that the chalk seemed to be formed mostly of the mineral or fossil remains of a ‘microscopic world’ and contained organisms of infinitesimal size, which he believed to be alive.

He also believed they might be of immense antiquity, as he had traced the block of limestone he had used to the Tertiary Period in geology; yet he found that stone cut from the solid ledge, with all air excluded, had ‘wonderful’ fermentative powers, which he traced to the same ‘little bodies’ that he had found to cause fermentation in his earlier experiments. He concluded that they must have lived embedded in the stone of the ledge for many thousands of years.

In 1866, he sent to the Academy of Science a memoir called On the Role of Chalk in Butyric and Lactic Fermentations, and the Living Organism Contained in it.3

In this paper, he named his ‘little bodies’ microzymas, from the Greek words for small ferment.

He also studied the relations of his microzymas of chalk to the molecular granulations of animal and vegetable cells, with many more geological examinations, and wrote a paper entitled On Geological Microzymas of Various Origins, which was abstracted in Comptes Rendus of the session of April 25, 1870.4

He proved that the molecular granulation found in yeast and other animal and vegetable cells had individuality and life, and also had the power to cause fermentation, and so he called them microzymas also.

He called his geological microzymas ‘morphologically identical’ with the microzymas of living beings.

In innumerable laboratory experiments, assisted now by Professor Estor – another very able scientist – he found microzymas everywhere, in all organic matter, in both healthy tissues and in diseased, where he also found them associated with various kinds of bacteria.

After painstaking study they decided that the microzymas rather than the cell were the elementary units of life, and were in fact the builders of cell tissues. They also concluded that bacteria are an outgrowth, or an evolutionary form, of microzymas that occur when a quantity of diseased tissues is broken up into its constituent elements.

In other words, all living organisms, he believed, from the one-celled amoeba to mankind, are associations of these minute living entities, and their presence is necessary for cell life to grow and for cells to be repaired.

Bacteria, they proved, can develop from microzyma by passing through certain intermediate stages, which they described, and which have been regarded by other researchers as different species!

The germs of the air, they decided, were merely microzymas, or bacteria set free when their former habitat was broken up, and they concluded that the ‘little bodies’ in the limestone and chalk were the survivors of living beings of long past ages.

This brought them to the beginning of 1868, and to test these ideas they buried the body of a kitten5 in pure carbonate of lime, specially prepared and creosoted to exclude any airborne or outside germs.

They placed it in a glass jar and covered the open top with several sheets of paper, placed so as to allow renewal of the air without allowing dust or organisms to enter. This was left on a shelf in Béchamp’s laboratory until the end of 1874.

When opened, it was found that the kitten’s body had been entirely consumed except for some small fragments of bone and dry matter. There was no smell, and the carbonate of lime was not discoloured.

Under the microscope, microzymas were not seen in the upper part of the carbonate of lime, but ‘swarmed by thousands’ in the part that had been below the kitten’s body.

As Béchamp thought that there might have been airborne germs in the kitten’s fur, lungs or intestines, he repeated this experiment, using the whole carcass of a kitten in one case, the liver only in another, and the heart, lungs and kidneys in a third test. These viscera were plunged into carbolic acid the moment they had been detached from the slaughtered animal. This experiment began in June 1875 and continued to August 1882 – over seven years.

It completely satisfied him that his idea – that microzymas were the living remains of plant and animal life of which, in either a recent or distant past, they had been the constituent cellular elements, and that they were in fact the primary anatomical elements of all living beings – was correct.

He proved that on the death of an organ its cells disappear, but the microzymas remain, imperishable.

As the geologists estimated that the chalk rocks or ledges from which he took his ‘geological microzymas’ were 11 million years old, it was proof positive that these microzymas could live in a dormant state for practically unlimited lengths of time.

When he again found bacteria in the remains of the second experiment, as he had in the first, he concluded that he had proved, because of the care taken to exclude airborne organisms, that bacteria can and do develop from microzymas, and are in fact a scavenging form of the microzymas, developed when death, decay, or disease cause an extraordinary amount of cell life either to need repair or be broken up.

He wrote in 1869:

“In typhoid fever, gangrene and anthrax, the existence has been found of bacteria in the tissues and blood, and one was very much disposed to take them for granted as cases of ordinary parasitism. It is evident, after what we have said, that instead of maintaining that the affection has had as its origin and cause the introduction into the organism of foreign germs with their consequent action, one should affirm that one only has to deal with an alteration of the function of microzymas, an alteration indicated by the change that has taken place in their form.”6

This view coincides well with the modern view of all germs found in nature, except those in the body, which are still looked on as causing the conditions they are found with, rather than being the result of these conditions, which is their true relation to them.

The Encyclopedia Britannica says in the entry on bacteriology:

“The common idea of bacteria in the minds of most people is that of a hidden and sinister scourge lying in wait for mankind. This popular conception is born of the fact that attention was first focused upon bacteria through the discovery, some 70 years ago, of the relationship of bacteria to disease in man, and that in its infancy the study of bacteriology was a branch of medical science.

“Relatively few people assign to bacteria the important position in the world of living things that they rightly occupy, for it is only a few of the bacteria known today that have developed in such a way that they can live in the human body, and for every one of this kind, there are scores of others which are perfectly harmless, and, far from being regarded as the enemies of mankind, must be numbered among his best friends.

“It is in fact no exaggeration to say that upon the activities of bacteria the very existence of man depends; indeed, without bacteria there could be no other living thing in the world; for every animal and plant owes its existence to the fertility of the soil and this in turn depends upon the activity of the micro-organisms which inhabit the soil in almost inconceivable numbers. It is one of the main objects of this article to show how true is this statement; there will be found in it only passing reference to the organisms which produce disease in man and animals; for information on these see Pathology and Immunity.”7

The writer of the above thoroughly understands germs or bacteria with only one exception; the bacteria found in man and animals do not cause disease. They have the same function as those found in the soil, or in sewage, or elsewhere in nature; they are there to rebuild dead or diseased tissues, or rework body wastes, and it is well known that they will not or cannot attack healthy tissues. They are as important and necessary to human life as those found elsewhere in nature, and are in reality just as harmless if we live correctly, as Béchamp clearly showed.

1. Annales de Chimie et du Physique, 4th series, 6, p246

2. Les Théorie du Microzyma, p.124

3. Comptes Rendus, 63, p.451 (session of Sept. 10th, 1866)

4. Ibid, 70, p.914

5. Les Microzymas, pp.626

6. Comptes Rendus, 75, p.1525 (session of Dec. 2nd, 1872)

7. Encyclopaedia Britannica, 14th ed., 2, p.899

Chapter 5 ♦ Silkworm Disease: Another Steal!

Between 1855 and 1865, a widespread epidemic among silkworms called pébrine alarmed the south of France, so much so that finally, in 1865, it drew national attention. Professor Béchamp, early in 1865, took up the study of this epidemic entirely at his own expense, and without the aid of others. He quickly found that it was caused by a small parasite.

His long experience with small micro-organisms, and the way creosote had inhibited their growth in his Beacon Experiment of 1854 and 1855, at once suggested the way out.

Hence he was able to state before the Agricultural Society of Herault the same year that pébrine was a parasitic disease and that thin creosote vapour would prevent the attack of the parasite.

However, in the meantime, the Government had taken an interest in the subject, and in June 1865 sent Pasteur down to investigate the disease.

Pasteur, with the prestige of being an official representative of the government, was able to centre all attention on his own work, to the depreciation of the work of others, though he admitted having never touched a silkworm before he started on this mission.

Nevertheless, the fact that something ‘official’ was being done caused agricultural societies to await his verdict, instead of at once taking up Professor Béchamp’s ideas.

Pasteur’s first statement on his new subject was made in September 1865, when he published a very erroneous description, claiming:

“The corpuscles are neither animal nor vegetable, but bodies more or less analogous to cancerous cells or those of pulmonary tuberculosis. From the point of view of a methodic classification, they should rather be ranged beside globules of pus, or globules of blood, or better still, granules of starch, than beside infusoria or moulds. … It is the chrysalide rather than the worm, that one should try to submit to proper remedies.”1

This description shows that he had no conception of the real nature of the problem.

Béchamp’s comment was:

“Thus this chemist, who is occupying himself with fermentation, has not begun to decide whether or not he is dealing with a ferment.”2

Pasteur, about this time, dropped his work because of the deaths of his father and two of his daughters, and before going back, spent a week at the Palace of Compiegne as the guest of Napoleon III.

In February 1866, he again took up the case of the silkworms and had the assistance this time of several able French scientists, yet they made very little progress on the problem.

Meanwhile, Béchamp had made further studies on pébrine, and sent a paper entitled On the Harmlessness of the Vapors of Creosote in the Rearing of Silkworms to the Academy of Science. In this article he repeated the statements he had made before the Agricultural Society at Herault and added that:

“The disease is parasitical. Pébrine attacks the worms at the start from the outside and the germ of the parasite comes from the air. The disease, in a word, is not primarily constitutional.”3

He described developing the eggs or seeds of the silkworm in an enclosure permeated with a slight odour of creosote, in which he produced eggs entirely free of pébrine, and it took so little creosote that his methods were commercially practical.

However, Pasteur had not yet found the true cause of the trouble. He sent a paper entitled New Studies on the Disease of Silkworms to the Academy, in which he said:

“I am very much inclined to believe that there is no actual disease of silkworms. I cannot better make clear my opinion of silkworm disease than by comparing it to the effects of pulmonary phthisis. My observations of this year have fortified me in the opinion that these little organisms are neither animalcules nor cryptogamic plants. It appears to me that it is chiefly the cellular tissue of all the organs that is transformed into corpuscles or produces them.”4

But again he guessed wrong, and neither he nor all of his assistants could prove statements that were false.

He also took a slap at Béchamp’s paper by saying:

“One would be tempted to believe, especially from the resemblance of the corpuscles to the spores of mucorina, that a parasite had invaded the nurseries. That would be an error.”

And yet Béchamp had already proved beyond question that it was nothing else but a parasite! Possibly, jealousy caused Pasteur to take a contrary view.

Pasteur, apparently, had not finally given up his ‘spontaneous generation’ ideas until 1862 or 1864, and since then, had ascribed all signs of fermentation, and all disease, to airborne germs, yet here he denies that this disease is parasitic! And after Béchamp’s papers proved it!

Béchamp answered him in a paper entitled Researches of the Nature of the Actual Disease of Silkworms which contained more proofs of its parasitical nature.

He said that the vibrant corpuscle:

“… is not a pathological production, something analogous to a globule of pus or a cancer cell, or to pulmonary tubercles, but is distinctly a cell of a vegetable nature.”5

In another paper6 Béchamp described experiments that proved the corpuscle to be an organized ferment that would invert sugar, and produce alcohol, acetic acid, etc.

This paper seemed to convince Pasteur that Béchamp was right, for in January 1867, in a letter written to Durny, the Minister of Public Instruction, he began to claim all credit for Béchamp’s ideas on the silkworm diseases.

Béchamp provided a still more complete account of his discovery which the Academy printed on April 29, 1867,7 and the same issue contained a letter from Pasteur to Dumas, dated April 24,8 in which he expressed regrets over his ‘mistakes’ and promised a paper with a complete story of the disease soon.

On May 13, 1867, Béchamp sent a letter to the Academy of Science pointing out Pasteur’s errors and asking for recognition of the priority of his own discoveries regarding silkworm diseases.9 He also sent another paper entitled New Facts to Help the History of the Actual Disease of Silkworms and the Nature of the Vibrant Corpuscles.10

In this paper he described the corpuscles as airborne and to be found on mulberry leaves, and he also described a second silkworm disease different from pébrine, which he called flacherie, and on which he had published a pamphlet privately, on April 11, 1867.

In the meantime he had also submitted several papers on various microscopic organisms, more or less broadening the general knowledge on this subject;11 one of these was a general study of bacterial development from his microzymas.12

In a paper entitled On the Microzymian Disease of Silkworms13 Béchamp gave a full description of this second disease called flacherie. This was published in the paper dated June 8, 1868, and on June 24 Pasteur wrote to Dumas claiming to have been the first to discover this second silkworm disease and demanding that a note he claimed to have sent to the Agricultural Society of Alais on June 1 be printed14 (as the records then contained no proof of Pasteur’s claim to this).

Béchamp answered this claim in a note entitled On the Microzymian Disease of Silkworms, in Regard to a Recent Communication of M. Pasteur,15 which was published under the date of July 13, 1867, in which he referred to his pamphlet of April 11, 1867, (revised and reprinted March 28, 1868) and his papers of May 13 and June 10, 1867, all of which were prior to any publication of Pasteur’s.

However, Pasteur used his prestige as a Government representative to brow-beat others into supporting him, and he was finally widely recognized, and Béchamp’s claims as to the discoveries on silkworm diseases were ignored. The majority of those who knew that his claims were false were afraid to oppose anyone who was so close to Napoleon, and who had so much official standing as Pasteur then had.

In his book on the diseases of silkworms,15 Pasteur takes all the credit for these discoveries, and shows how ignorant of the subject he still is by ridiculing Béchamp’s statements that creosote was a preventative – so he knew of them!

Ethel Hume says that members of the Academy actually asked Professor Béchamp to drop his use of the word microzyma, and even to drop his work!

In Microbe Hunters, Paul de Kruif gives a slightly different version of Pasteur’s work on silkworms from that outlined above. He states that Dumas, his old professor, appealed to Pasteur to help the silkworm growers of southern France, and continues:

“Anything but a respecter of persons, Pasteur, who loved and respected himself above all men, had always kept a touching reverence for Dumas. He must help his sad old professor! But how? It is doubtful at this time that Pasteur could have told a silkworm from an angle worm! Indeed, when he was first given a cocoon to examine, he held it up to his ear, shook it and cried:

‘Why there is something inside it!’” (p.91.)

De Kruif also ascribes the belated discovery that pébrine was a parasitical disease to Gernez, one of his assistants:

“Gernez hurried to Pasteur. ‘It is solved,’ he cried, ‘the little globules are alive – they are parasites! They are what makes the worms sick!’

“It was six months before Pasteur was convinced that Gernez was right, but when at last he understood, he swooped back to his work, and once more called the committee together:

‘The little corpuscles are not only a sign of the disease, they are its cause. These globules are alive, they multiply, they force themselves into every part of the moth’s body.’” (p.95.)

It is strange that with the dispute raging between Béchamp and Pasteur over who had discovered that pébrine was a parasitical disease, Gernez did not speak of his own claims in the matter – perhaps a job was more important.

De Kruif continues:

“He was 45. He wallowed in this glory for a moment and then – having saved the silkworm industry with the help of God and Gernez – he raised his eyes toward one of those bright, impossible, but always partly true visions that it was his poet’s gift to see. He raised his artist’s eyes from the sickness of silkworms to the sorrows of mankind:

‘It is in the power of man to make parasitic maladies disappear from the face of the globe, if the doctrine of spontaneous generation is wrong as I am sure it is!’” (p.97.)

His 45th year must have been 1867, and Béchamp had proven spontaneous generation wrong in 1855 or ’56, as described earlier – at least 10 years beforehand.

Clearly de Kruif did not look far enough; the name of Béchamp – the only ‘microbe hunter’ who really understood their true place in nature – does not appear in his book Microbe Hunters at all!

In spite of all his errors in the work on silkworms, and because of his high position and royal favouritism, Pasteur was put in charge of the practical measures of fighting this parasite, and of course did not adopt Béchamp’s method of using creosote vapour.

Dr A. Lateud, at one time editor of the Journal de Médecine de Paris, charged that whereas in 1850 France had produced 30 million kilograms of cocoons, its output had sunk to 15 million kilograms in 1866-7 due to the epidemic. After Pasteur’s methods of ‘prevention’ had been introduced, production shrank to 8 million kilograms in 1873 and as low as 2 million kilograms in subsequent years. He continued:

“That is the way in which Pasteur saved sericulture! The reputation which he still preserves in this respect among ignoramuses and short-sighted savants has been brought into being:

  • “by himself, by means of his inaccurate assertions;
  • “by the sellers of microscopic seeds based on on the Pasteur system, who have realized big benefits at the expense of the cultivators;
  • “by the complicity of the Academies and public bodies, which, without any investigation, reply to the complaints of the cultivators: ‘But sericulture is saved! Make use of Pasteur’s system!’ However, not everybody is inclined to employ a system that consists in enriching oneself by the ruination of others.”17

Plainly his sins found him out here – at least with those who were in closest touch with the silkworm cultivators!

It is astonishing, in view of such a failure – and after Béchamp had shown how to prevent these diseases – that Pasteur’s reputation did not go down in a public scandal!

Apparently royal favour and the Academies and public bodies protected him from this.

1. Comptes Rendus, 61, p.506

2. Les Grand Problémes Medicaux, p.7

3. Comptes Rendus, 62, p.1341 (session of June 18 1866)

4. ibid, 63, pp.126-142 (session of July 23 1866)

5. ibid, 63, p.311 (session of August 13, 1866)

6. ibid, 63, p.391 (session of August 27, 1866)

7. ibid, 64, p.873 (session of April 29, 1867)

8. ibid, 64, p.835 (session of April 29, 1867)

9. ibid, 64, p.1042 (session of April 29 1867)

10. ibid, 64, p.1043 (session of April 29 1867)

11. ibid, 64, p.696 and 66, pp.366 and 421

12. ibid, 66, p.859 (session of May 4 1868)

13. ibid, 66, p.1160 (session of June 8 1868)

14. ibid, 66, p.1289 (session of June 29 1868)

15. ibid, 67, p.102 (session of July 13 1868)

16. Studies sur le Maladie des Vers-a-sois, 1870

17. Etudes sur la Rage, pp.427-8

Chapter 6 ♦ Pasteur also a Faker: Antisepsis

While many of Pasteur’s contemporaries must have known of his plagiarisms of Béchamp’s work, they were cowed into silence, or kept out of the press by Pasteur’s bullying tactics, as well as by his prestige, not only in the public eye and with royalty, but also with the ‘Academies and public bodies’ Dr Lateud refers to.

Ethel Hume goes on to show that his treatment for rabies and his anthrax serum were the same colossal failure and fraud, as will be shown in Chapter 8, and she discusses other plagiarisms on Pasteur’s part, but it hardly seems necessary to go into all of these matters here. We have seen enough evidence of incompetence and fraud to forever doubt any further statements that bear his signature – but there is one more piece of work that is worth looking into.

Some years after the events we have described, Dr M. L. Leverson, an American physician, discovered some of Professor Béchamp’s writings in New York and immediately realized that they anticipated Pasteur in certain important points. He went to France, met Professor Béchamp, and heard the story of the plagiarism from him, after which he did a great deal to bring Béchamp’s work to public attention.

He was one of the first in the United States to recognize Béchamp’s priority in regard to most of the discoveries generally credited to Pasteur, and in a lecture entitled Pasteur, the Plagiarist, delivered at Claridges Hotel, London, on May 25, 1911, outlined briefly Béchamp’s claim to priority, and added the charge that Pasteur had deliberately faked an important paper!

He said in part:

“Pasteur’s plagiarisms of the discoveries of Béchamp – and of Béchamp’s collaborators – run through the whole of Pasteur’s life and work, except as to crystallography, which may or may not have been his own. I have not investigated that part of his career, nor do I feel any interest in it.”

“The tracings of some of these plagiarisms, though they can be clearly demonstrated, are yet somewhat intricate, too much so for this paper; but there is one involving the claim by Pasteur to have discovered the cause of one of the diseased conditions which assail the silkworm, which can be verified by anyone able to read the French language. It is the following…”

After then describing some of the material we have covered in Chapter 5, he continues:

“But I have a still graver and more startling charge to bring against Pasteur as a supposed man of science.”

Scientific Bluff

“Finding how readily the ‘men of science’ of his day accepted his fairy tales, in a voluminous memoir of no value (published in the Annales de Chimie et de Physique 3rd series., Vol. LVIII), is to be found on page 381 a section entitled Production of Yeast in a Medium Formed of Sugar, of a Salt of Ammonia and of Phosphates.

“The real, though not confessed, object of the paper was to cause it to be believed that he, and not Béchamp, was the first to produce a ferment in a fermentative medium without albuminoid matter.

“However, the alleged experiment described in the memoir was a fake – purely and simply a fake. Yeast cannot be produced under the conditions of that section! If those of my hearers or any other physician having some knowledge of physiological chemistry will take the pains to read this section of Pasteur’s memoir with attention, he will see for himself that yeast cannot be so produced, and he can prove it by reproducing the experiment as described.

“Now mark what – supposing I am right in this – this memoir does prove. It proves that Pasteur was so ignorant of physiological chemistry that he believed yeast could be so produced, or else he was so confident of the ignorant confidence of the medical profession in himself, that he believed he could bluff it through. In this last belief, he was correct for a time. I can only hope that the exposure I am making of Pasteur’s ignorance and dishonesty will lead to a serious overhauling of all his work.

“It was Béchamp who discovered and expounded the theory of antisepsis which Pasteur permitted to be ascribed to himself. In his Studies on Fermentation, Pasteur published a letter from Lord Lister, then Mr. Surgeon Lister, in which he claims that he learned the principles of antisepsis from Pasteur. I do not doubt this statement of the noble Lord, for besides accepting Mr. Lister as a gentleman of veracity, I will give you an additional reason for accepting that statement.”

Lister’s Blunder

“When Mr Lister began his antiseptic operations, they were generally successful, but a few days later his patients succumbed to carbolic acid or mercuric poisoning, so that it became a gruesome medical joke to say that ‘the operation was successful, but the patient died’.

“Now Mr Lister, though a very skilled surgeon and, I believe, having great powers of observation, had established the technique of his operations upon the teachings of a man who had plagiarized the discovery without understanding the principle upon which it was based. Not unnaturally, Lister used doses of carbolic acid, which, when placed upon an open wound or respired by a patient, were lethal.

“But, thanks to his careful observations, he gradually reduced the quantity of carbolic acid or sublimate of mercury employed, until at last ‘his operations were successful and the patients lived’, as they would have done from the beginning, had he obtained his knowledge of the principles of antisepsis from their discoverer, Béchamp – who had warned against the use of any but a very minute dose of carbolic acid – instead of from the plagiarist Pasteur, who did not know why the dose should be so limited.

“From the outline I have now given you, you may form some idea of the ignorance of the man who, for more than thirty years, official medicine has been worshipping as a god. But this is only a small part of the mischief perpetrated. Instead of making progress in therapeutics during the past thirty or forty years, medicine – outside of surgery – has fearfully retrograded, and the medical profession today is, in my judgment, in a more degraded condition than ever before in its history.

“I know that at first your minds will rebel against this statement, but some facts will prove to every mind possessed of common sense that it is true.”

The Danger of Inoculating

After discussing the practice of medicine in the past and saying that since Jenner’s and Pasteur’s days the modern effort is to make the sick well, he says of inoculations:

“When a drug is administered by the mouth, as was beautifully pointed out by Dr J. Garth Wilkinson, in proceeding along the alimentary canal it encounters along its whole line a series of chemical laboratories, wherein it is analysed, synthesized, and deleterious matter prepared for excretion, and finally excreted, or it may be ejected from the stomach, or overcome by an antidote.

“But when nature’s coat of mail, the skin, is violated, and the drug inserted beneath the skin, nature’s line of defence is outflanked, and rarely can anything be done to hinder or prevent the action of the drug, no matter how injurious – or even fatal – it may be. All the physicians of the world are incompetent either to foresee its action or to hinder it.

“Even pure water has been known to act as a violent and foudroyant poison when injected into the blood stream. How much more dangerous is it, then, to inject poisons known to be such, whether modified in the fanciful manner at present fashionable among vivisectionists or in any other manner. These simple considerations show that inoculation should be regarded as malpractice to be tolerated only in case of extreme danger where the educated physician sees no other chance of saving life.”

The Germ Theory Fetish

“Now the forcing of these inoculations upon individuals by law is one of the worst tyrannies imaginable, and should be resisted, even to the death of the official who is enforcing it. English-speaking people need to have ideals of liberty refreshed by a study of the history of Wat Tyler, who headed one of the most justifiable rebellions in history, and although treacherously murdered by the then Lord Mayor of London, his example should be held up to all our children for imitation…

“The entire fabric of the germ theory of disease rests upon assumptions which not only have not been proved, but which are incapable of proof, and many of them can be proved to be the reverse of truth. The basic one of these unproven assumptions, the credit for which in its present form is wholly due to Pasteur, is the hypothesis that all the so-called infectious and contagious disorders are caused by germs, each disease having its own specific germ, these germs having existed in the air from the beginning of things, and that though the body is closed to these pathogens’ germs when in good health, when the vitality is lowered the body becomes susceptible to their inroads.”

I agree most heartily with Dr Leverson’s statement that

“the forcing of these inoculations upon individuals by law is one of the worst tyrannies imaginable, and should be resisted even to the death of the official who is enforcing it.”

Strong words, but absolutely right!

Professor F. W. Newman of Oxford University has said:

“Against the body of a healthy man, Parliament has no right of assault whatever under pretence of the public health; nor any the more against the body of a healthy infant. To forbid perfect health is a tyrannical wickedness, just as much as to forbid chastity or sobriety. No lawgiver can have the right. The law is an unendurable usurpation, and creates the right of resistance.”

And Blackstone says:

“No laws are binding upon the human subject which assault the body or violate the conscience.”

In the case of the Union Pacific Railway vs Botsford, the United States Supreme Court said:

“… no right is held more sacred or is more carefully guarded by the common law than the right of every individual to the possession and control of his own person, free from all restraint or interference of others, unless by clear and unquestioned authority of law.

“As well said by Judge Cooley:

‘The right of one’s person may be said to be a right of complete immunity; to be let alone.’(Cooley on Torts 29)

“The inviolability of the person is as much invaded by a compulsory stripping as by a blow. To compel anyone, and especially a woman, to lay bare the body or to submit it to the touch of a stranger, without lawful authority, is an indignity, an assault, and a trespass.”(141 U.S. 250)

In 1903, Judge Woodward of the New York Appellate Court said in the Viemeister case:

“It may be conceded that the legislature has no constitutional right to compel any person to vaccination.”(84 N.Y. Supp. 712)

In the Supreme Court, Columbia County, N.Y., in 1910, Judge Le Boeuf, in the second trial of the Bolinger case, instructed the jury as follows:

“Now I have charged you that the assault which is claimed to have existed here due to the forcible vaccination, that is, if it was against this man’s will, is one which you must consider. And the reason of that is: This man, in the eyes of the law, just as you and I and all of us in this courtroom, has the right to be let alone. We all have the right to the freedom of our persons and that freedom of our persons may not be unlawfully invaded. That is a great right. It is one of the most important rights we have.”

I believe these quotations from court documents indicate clearly that anyone has a right to protect himself or his family from the pus-squirters of the A.M.A.

Over 60 years ago [in 1942, tlz.] the famous English physician, Dr Charles Creighton, said in Jenner and Vaccination (1879):

“The anti-vaccinationists have knocked the bottom out of a grotesque superstition.”

However, it has been revived, and needs some more ‘knocks’.

The doctors will not willingly give up such a lucrative practice as the use of biologicals, and so parents and the public must do something to stop this blood-poisoning. What, then, can be done?

I have seen a little girl, upon being vaccinated (or ‘inoculated’), go to school, promptly develop ‘leaky heart valves’ and die of ‘heart trouble’ about two years later, hardly ten years old. I don’t believe that either her parents, schoolmates, or teacher, or even the doctor concerned, saw any connection between the vaccination, or inoculation, and the leaky heart valves – but there was a connection – see my pamphlet The So-called Biologicals have Created a New Form of Heart Disease.

And thousands of such deaths are caused every year. What are we going to do to stop it?

In the whole history of mankind, the only adequate answer to tyranny that humanity has ever had has been the overthrow of the tyrant; and the A.M.A. and their cronies have certainly been tyrannical in their efforts to sell their decayed animal-pus biologicals for many years. I believe that if these efforts at compulsion, coercion or compulsory laws to force the use of any kind of biological or so-called ‘tests’ of any kind are pushed much further, they will lead to trouble.

As we show in this book, the underlying ‘germ theory’ is a fraud, and everything based on it is also fraudulent, and should be forbidden by law; and when the public fully realizes what a colossal fraud the use of these decayed animal-pus concoctions is, you won’t even be able to jail a man for shooting a pus-doctor who tries to vaccinate, inoculate, or ‘test’ his children.

We will outline, further on, a safe method of controlling infections.

Dr Leverson goes on to describe disease as nature’s attempt to eliminate waste, and diseased tissues as being due to improper living; and suggests plenty of fresh air, the best of sanitation, very scanty clothes such as gymnasium costumes for everyday use, and a scientific study of diet; he believes overeating causes ‘an enormous number of diseased conditions’.

All of these ideas would undoubtedly lead to better health and longer life than can be obtained through serology.

It is now over 30 years since Dr Leverson expressed the hope that his exposure would lead to a serious overhauling of Pasteur’s work, and it should be done by someone who understands physiological chemistry.

I feel as he seems to – that the allopathic mind is hardly to be trusted with such important work!

Chapter 7 ♦ Are Biologicals Injurious?

The 11th Report of the Medical Officer of the Privy Council of England (1868) contains a paper by Dr Burdon Sanderson entitled: On the Inoculability and Development of Tubercles (p.91). In this paper he describes experiments he made which proved to his satisfaction that tuberculosis often followed the inoculation of animals with various materials (mostly biological) from non-tubercular sources, and that even a wound might be followed by tuberculosis. He says in part (p.92):

“The facts from which I had concluded that tuberculosis may originate traumatically, although very limited in number, were so positive in nature that I ventured to state that the results of tuberculosis inoculation could be no longer regarded as necessarily dependent on any property or action possessed by the inoculated material in virtue of its having been taken from a tuberculous individual.

“The truth of this inference has now been established by the experiments of two competent observers, Dr Wilson Fox, Professor of Clinical Medicine in University College1 and Dr Cohnheim of Berlin. The following paragraph contains a summary of their results, which are the more valuable as they were arrived at altogether independently and without knowledge either of each other’s inquiries or mine.

“From the tabular summary of Dr Fox’s experiments (117 in number) it appears that of 70 animals inoculated with various products derived from the bodies of non-tuberculous patients, about half (34) became tuberculous. In addition, five animals were inoculated with putrid but originally healthy muscle, and four of them became tuberculous, as was found when they were killed at various periods from 84 to 122 days after inoculation. Of seven animals in which setons or other mechanical irritants were introduced under the skin, two became tuberculous.2

“This research, no less remarkable for the accuracy and completeness of the anatomical details, than for the conclusiveness of the experiments, was followed only the other day by another in Berlin, which although of similar nature, appears by internal evidence to have been conducted in entire ignorance of the fact that several of the questions investigated had already been completely settled in England.

Drs Cohnheim and Frankel, to establish whether artificial tubercle owe its origin to a specific virus3 introduced into the peritonaeal cavities of guinea pigs portions of various tumours (carcinoma, sarcoma, condyloma, etc.) as well as portions of healthy but partly decomposed tissue. Subsequently they employed in the same way a variety of insoluble inert substances such as blotting paper, charpie, gutta percha, caoutchouc, vulcanite, etc. In those animals that survived the immediate effects of the injury, emaciation supervened sooner or later and the animal eventually died with tuberculosis of the peritoneum, liver, spleen, lungs, and other organs, the morbid appearances corresponding in every respect with those described in my last report.

“As regards the bearing of these facts on the general question of the nature and origin of tuberculosis; I concluded from my own observations that there is no structural distinction between the artificial disease and human tubercle, so long as the term is confined, as all accurate writers are now accustomed to confine it, to miliary tuberculosis; but I considered it necessary to maintain a reserve as to its relation with the many pathological processes which are spoken of as tuberculosis in the common language of practical medicine and surgery. In going so far the two pathologists already quoted have fully agreed with me.

“Dr Fox says:”

‘I must confess that sceptical as everyone must naturally at first feel on this subject, the cumulative force of the evidence in favour of the tubercular nature of these growths appears to me irresistible. We are either dealing with tubercle, or we have before us a new and hitherto unknown constitutional disease of the rodentia, consisting of growths which, to the naked eye and in their histology, correspond with all the essential features of tubercle in man; which occur not only in the organs which are the chosen seats of tubercle in man, but also in the same parts of those organs; which have the same vital characters, and the same early degenerative cheesy changes, not suppuration nor acute softening, and with no marked characters sufficient to distinguish them from tubercle.4

“Cohnheim says:

‘All the marks by which tubercle is characterized are present; the agreement of the product of inoculation with human miliary tubercle could not be more complete than it is, whether regard be had to its extended distribution and to the great variety of organs affected, (peritoneum, pleura, lungs, liver, spleen, lymphatic glands, and even the choroid), or to its macroscopic and microscopic characters.’”

Gould, in his Pocket Cyclopædia of Medicine and Surgery describes ‘acute miliary tuberculosis’ as:

“An acute and rapid form of tuberculosis, which generally occurs in persons under 15 years of age, and in which the tubercle bacilli are rapidly disseminated through the body by the breaking down of some localized form of the disease … the duration is from 2 to 4 weeks and the termination is fatal.”

Or, could not this ‘localized form’ be introduced by a needle, in the way Dr Sanderson describes? Are not ‘persons under 15’ the school doctor’s best customers for their so-called biologicals? And does not this ‘rapid dissemination through the body’ sound remarkably like de Kruif’s description of the way in which Koch’s tuberculous germs spread through his guinea pigs?

Ethel Hume says in Béchamp or Pasteur?:

“It is noteworthy that neither Pasteur nor any of his successors have ever induced a complaint by the inoculation of air-carried bacteria, but only by injections from bodily sources.”

I believe this would account for a very large part of our ‘miliary tuberculosis’ in persons under 15; undoubtedly it followed the injection of some biological! And Ethel Hume’s description would include all biologicals of every type!

Dr Sanderson continues:

“My further inquiries lead me to believe, in the first place that these characters belong much more generally to tuberculous growths than I had at first supposed; and secondly, that those normal tissues which possess them are much more liable to become the seat of the tuberculous process than others.”

This is probably the most striking evidence in print that almost any sort of inoculation can cause tuberculosis in the animal inoculated, and of course it is reasonable to deduce from this that the same non-tuberculous inoculations would cause tuberculosis in man, any man, and in all probability, from any biological product whatsoever! Yet the serum doctors will tell us that these products are perfectly harmless!

Tuberculin a fraud

The above article, which from the day it was first printed should have forever stopped the use of all biologicals on humans, was published over 20 years before Robert Koch of Berlin brought out his Tuberculin (in 1890), which proved such a terrible failure!

The Zoophilist for May 1st 1891 reported deaths in 123 ‘selected’ cases in Berlin from November 1890 to February 1891 which caused Koch to fall ‘under a cloud’, but he did not give up until the government finally closed him down because of the terrible death rate!

Dr Paul de Kruif describes this work of Koch’s on the tuberculosis germ in rather lurid language,5 yet recent efforts to produce a serum for tuberculosis seem to justify his words. He says of Koch’s search for the microbe:

“‘I have it!’ he whispered, and called the busy Loeffler and the faithful Gaffhy from their own spyings on other microbes.”

“‘Look,’ Koch cried, ‘one little speck of tubercle I put into this beast six weeks ago – there could not have been more than a few hundred of those bacilli in that small bit – now they’ve grown into billions! What devils they are, those germs – from that one place in the guinea pig’s groin they have sneaked everywhere into his body, they have gnawed, they have gone through the walls of his arteries … the blood has carried them into his bones … into the farthest corner of his brain…’”

Read that over when your child brings home a card from school requesting permission to put the same sort of stuff into his blood, and tear up the card! He says that Koch found and grew 43 different families or varieties of these deadly germs. I believe that by the doctors’ standards at least, this would necessitate 43 different serums to immunize one against all 43 families, and this is probably not all the varieties there are of tuberculosis germs alone.

However, de Kruif passes over tuberculin with astonishing brevity, considering the space given to other matters that were of less importance. He says apologetically:

“… he was enormously respected, and against his own judgement he was trying to convince himself he had discovered a cure for tuberculosis. The authorities (scientists have reason occasionally to curse all authorities, no matter how benevolent) were putting pressure on him. At least so it is whispered now by veteran microbe hunters who were there and remember those brave times.

‘We have showered you with medals and microscopes and guinea pigs – take a chance now and give us a big cure, for the glory of the fatherland, as Pasteur has done for the glory of France!’

“It was ominous stuff like this that Koch was always hearing. He listened at last, and who can blame him, for what man can remain at his proper business of finding out the ways of microbes with governments bawling for a place in the sun – or with mothers calling? So Koch listened and prepared his own disaster by telling the world about his Tuberculin.”6

And here de Kruif changes the subject very abruptly! On page 299 he refers to it again, in discussing malaria, as follows:

“Dean of the microbe hunters of the world, Tsar of Science (his crown was only a little battered) Koch had come to Italy to prove that mosquitoes carry malaria from man to man.

“Koch was an extremely grumpy, quiet, and restless man now; sad because of the affair of his consumption cure (which had killed a considerable number of people) … so Koch went from one end of the world to the other, offering to conquer plagues but not quite succeeding.”

Neither are his successes in the use of serums, nor is there any likelihood of success in that direction, as we hope to show.

J.W. Browne, Medical Superindent of the Kalyra Sanatorium, South Australia, quotes Koch at length to the effect that, while an injection of tuberculin into a healthy person will probably start a tubercular sore, an injection into anyone already infected will counteract or ‘kill’ the first infection, without doing anything more!

Note that he admits that it causes tubercular sores in a healthy individual! Hence you’d better know whether you have tuberculosis or not before you take it!

However, this reversible characteristic of making the well sick, and the sick well, existed only in Koch’s imagination, as is indicated in his own work. Anyone with such a belief must be credited with care in giving such stuff only to tubercular people, and those who received it died so fast the government had to shut him down! Incidentally, cattlemen have contended for many years that it made healthy cattle tubercular.

Dr Browne says:

“To date, upwards of two hundred different forms of tuberculin have been prepared and described.

“The simple fact of the matter is that no one has yet been able to repeat Koch’s experiment successfully. There is no evidence but Koch’s in favour of tuberculin as a therapeutic cure for tuberculosis in guinea pigs, in calves, or in man. No one but Koch has been able to cure an infected guinea pig by the use of tuberculin of any sort.

“Koch, as Shera says, was an optimist. There is no question that tuberculin can do infinite harm. Scores of people have died prematurely at its hands. Never was there such a commercial vaccine as this one, and never has there been such a gigantic hoax. Tuberculin, Shera says, should not come within the range of vaccine therapy. Whatever good results are imputed to tuberculin must have occurred in spite of it, for its virtues are founded on experiments which cannot be repeated.

“The disbeliever too, can point to many cases where the administration of tuberculin in pulmonary disease has been undoubtedly followed by disaster and, while he freely admits the undoubted powers of the tuberculin therapist to stir up the embers and kindle the fire, he has hitherto asked him in vain for any evidence of power to extinguish the fire.”

He (rightly, I believe) considers pulmonary tuberculosis to be at least in part ‘and to a greater or less extent’ a septicemia, and adds:

“The failure of vaccines to affect the disease in any but an adverse manner is thus explained. As we all know, vaccines have invariably been found useless or worse than useless in septicemias.”7

Such statements, coming from a physician of Dr Browne’s experience, should write finis on the use of tuberculin as a cure forever; and it is no better as a ‘test’.

Drs Petroff and Branch, in a discussion of the B.C.G. vaccine used on children, finds that tuberculin seems to spread tuberculosis in those who have the latent or ‘benign’ form which vaccination is supposed to give.

Note also that the tuberculin seemed to spread tuberculosis in these cattle ‘tests’ as it did in Koch’s experiments on humans. They say:

“Tzekhnovitzer claims that guinea pigs become hypersensitive to tuberculin after treatment with B.C.G … 70% of those infected orally and 45% of those infected by the subcutaneous route react.”

Immunity in animals vaccinated with B.C.G.

“Guerin, Richart and Bossiera studied a large number of cattle on a farm. On this farm in 1915 in a herd of 67 head, 47% reacted positively to the tuberculin test. Year after year, the positive animals were slaughtered. In 1918, 38% were still positive to the tuberculin test. In 1920, the number of reactors was 41.7%.

“Vaccination in the newborn cattle started on Jan. 1, 1921. In 1922, one year after the vaccination, 20 cattle gave a definitely positive and nine a very suspicious tuberculin reaction, or a total of 45% of 64 head. Many of these animals were vaccinated and revaccinated. In 1923 there remained 26 of the 1919-1920 year animals, all giving a positive tuberculin reaction.”

Note that after 47% were slaughtered in 1915, as were all animals testing positive in the following years, 38% were tubercular in 1918, and a full 100% of those animals which remained from the 1919–20 vaccinated group all gave a positive ‘test’. This was undoubtedly due either to the vaccines used or the ‘tests’ themselves, which confirms the opinions of the authorities quoted above! Could any dairyman survive such a loss?

They continue:

“In the meantime, the second generation of these vaccinated animals were revaccinated, and the vaccination repeated each following year. There is no record of how many of the vaccinated cattle became infected, as the tuberculin test was omitted on Calmettes’ suggestion, as he believes it to be of doubtful value, giving no information as far as exogenous (outside) infection is concerned.

Furthermore if in the vaccinated cattle an implantation of virulent organisms has taken place, setting up only a benign tuberculosis, tuberculin administered may bring about a violent allergic reaction disseminating the virulent organisms. In such an event, progressive disease may follow

“Gradually the animal becomes resistant to this particular organism. However, as soon as a new organism is introduced into the herd, the occurrence of the disease is much more marked than before.”8

They do not mention the fact that these ‘implantations’ may also occur in your child; nor do they realise that they can come through a change of the germ in the vaccine, as I showed in Germ Mutation (now out of print).

As occurred with ‘flu’ in the war, which was merely a mutation of the typhoid germ in the vaccines used against typhoid and paratyphoid, every vaccine may produce a ‘new’ form of germ which, as noted above, may ‘make the occurrence of the disease much more marked than previously’.

This is why we had the 1918 flu epidemic, with the highest death rate on record. It is the reason Koch had so many deaths, and also the reason for the large increases in the death rates of other diseases as noted in Chapter 9.

Koch found 43 varieties or strains of tuberculosis and there are probably as many strains of any other disease. The very multiplicity of these strains, and the ease with which modification can occur on the shelf or in the tissues, is the fundamental reason why biologicals can never be used successfully.

F. Loehnis, soil biologist, and N. R. Smith of the U.S. Department of Agriculture have discussed this variability of germs at considerable length and conclude that any germ can break down into a filterable fluid and then develop into new forms that may be radically different from the original germ, their new characteristics depending mostly upon their environment. They believe this change is constantly going on in all groups of germs.9

Hence new strains are always being formed and are often more virulent than the old.

Doctors Petroff and Branch add:

“It seems that in spite of the vaccinations with B.C.G., and the sociological measures, the implantation with violent tubercle has taken place…

“Lakhms of Lithuania, studying 472 vaccinated infants, reports that he obtained 10 times more positive reactions in the vaccinated children than in the unvaccinated.”

The real fact is that tuberculin never had any diagnostic value. It was not offered as a test on animals until its failure as a cure on humans caused the German government to forbid such use; in other words, the manufacturers ‘discovered’ or invented this new use for it to preserve a market. The ‘test’ on cattle circumvented both the prohibition and its ill-repute as a cure, thus continuing the profits, which is all it is good for.

Read the account of the United States Agricultural Department’s ‘tests’ on animals infected with the hoof-and-mouth disease from vaccines, in Chapter 8.

In Fasting and Man’s Correct Diet, The Tuberculin Test a Fraud (out of print), Immunity (also out of print), and Drugless Cures, I give additional evidence that the use of tuberculin was a fraud, utterly useless, and that more recent serums are no better.

Biologicals may dissolve the red blood corpuscles

It has also been found that the soluble ferments of many animal serums will, in some humans at least, dissolve the red blood corpuscles.

Elie Metchnikoff, the famous Russian scientist, says:

“It has long been known, however, that the serum of the blood of many animals will destroy the red corpuscles of a different species. This demonstration was afforded during the period when attempts were being made to transfuse the defibrinated blood of mammals, especially of the sheep, into man. This practice had to be abandoned in consequence of the difficulties resulting from the solution of the human red corpuscles.10

“Later, Buchner11 compared the action of alexine (the name given to the substance found to cause this action) to that of soluble ferments and referred it to the category of the digestive diastases.”

This alexine is probably the same thing described by Béchamp as the liquid ferment mentioned in Chapter 2, and it should not destroy or even injure perfectly healthy blood or tissues, but who is perfectly healthy?

Dr Leverson says in the preface to his translation of The Blood and its Third Anatomical Element that Béchamp isolated a series of soluble ferments which he called zymases, but which plagiarists renamed diastases to obscure his discoveries. Likewise, Béchamp discovered the reason for the coagulation of the blood.

Metchnikoff continues:

“According to him the same alexine is capable of dissolving the red blood corpuscles of several species of vertebrates. Bordet,12 in a series of researches made in the Pasteur Institute, confirmed this view. He came to the conclusion that the alexines of the various species of animals differ from one another. Thus the alexine of the blood serum of the rabbit is not the same as that found in the serum of the guinea-pig or dog. Nevertheless each of these alexines is capable of exerting a solvent action on the red blood corpuscles of several species.”

He continues, on page 95:

“It may, however, be admitted that the action of alexine (complement) comes under the category of phenomena that are produced by soluble ferments. The substance which dissolves the red blood corpuscles of mammals or a portion only of those of birds, undoubtedly presents great analogies to the digestive ferments. As has been mentioned repeatedly, it is very sensitive to the action of heat and is completely destroyed by heating for one hour at 55°C. In this respect, it closely resembles the macrocytase of macrophagic organs which also dissolves red corpuscles. As it is the macrophages which ingest and digest the red blood corpuscles in the organism, it is evident that alexine is nothing but the macrocytase which has escaped from the phagocytes during the preparation of the serums.”

On page 401 of the same book, discussing artificial immunity against toxins rather than microbes, he says:

“When micro-organisms, living or dead, are introduced into an animal, it is found that anti-toxins do not as a rule, appear in the fluids; in these cases, the reaction is set up mainly by the microphages. The microphages represent the principal source of anti-toxins.”

Is this point clear? All animal blood serums can dissolve the red blood corpuscles of several other species of animals, and many of them, for example that of the sheep, can dissolve the red blood corpuscles of man!

It is also possible that due to the wide variations in the character of the blood and blood serum, etc., both in the animals used and in the patients treated, due to both individual and possibly also racial differences, the serum from any particular animal might have a very injurious effect on the blood or other body fluids of a percentage of human patients treated, as indicated by the many deaths that follow the use of anti-toxin, even though it might not be injurious to all.

Note that they compare this stuff to a soluble ferment, which can go through a china filter, and eat red blood corpuscles, pink dynamite and other things; and this is ‘the principal source of anti-toxins’.

It may be true that most horses’ blood serum will not dissolve human red blood corpuscles, but how can we know, with all the variations possible, both in the horse, and in man, that some particular horse serum will not dissolve the red blood corpuscles of one or more children in any school which the serum squirters choose to ‘protect’, as they call it?

This might be the direct cause of the tuberculosis discussed above, and many other troubles that often follow the vaccination of thousands of children, and others.

We quoted Professor Béchamp as to the amount of material a solvent ferment can digest in Chapter 2, and Béchamp and other authorities say that a solvent ferment will survive much higher temperatures than 55°C. This danger, therefore, exists in almost every biological on the market!

There is also the danger that some serum might contain the alexine of some animal other than a horse, which could be even more dangerous.

Furthermore, even though a serum cannot dissolve the red blood corpuscles, it might dissolve the leucocytes, the so-called white corpuscles, and this tendency seems to be much more common; in fact, it seems to be the basis of the process of artificial immunity!

For instance, Metchnikoff says:

“When into the peritonaeal cavity of vaccinated guinea-pigs a certain quantity of cholera culture containing virulent and very motile vibrios is injected, we find that in the peritonaeal fluid drawn off by means of a fine pipette, the vibrios have undergone profound changes in the refractory organism. Even a few minutes after the injection of the vibrios, the leucocytes disappear almost completely from the peritonaeal fluid; and only a few small lymphocytes and a large number of vibrios, the majority of which are already transformed into granules, are found; and there is presented a most typical case of Pfeiffer’s phenomenon.

“Alongside the round granules may be seen swollen vibrios, and others which have kept their normal form, but all are absolutely motionless. Some of these granules are gathered into small clumps, others remain isolated in the fluid. When to the hanging drop containing these transformed vibrios a small quantity of a dilute aqueous solution of methylene blue is added, we observe that certain granules stain very deeply, while others take on merely a very pale tint, scarcely visible. Many of these granules are still alive, because it is easy to watch them develop outside the animal and elongate into new vibrios. A large number of the granules, however, no longer exhibit any signs of life and are evidently dead.

“R. Pfeiffer and certain other observers affirm that the granules may be completely dissolved in the peritonaeal fluid just as a piece of sugar dissolves in water. We have repeatedly sought for this disappearance of the granules in hanging drops of the peritonaeal fluid, without being able to find any diminution in the number of these transformed vibrios, even after several days. Nor have we been able to observe the phenomenon of the solution of the granules. It is, at any rate, indisputable that this granular transformation is a manifestation of very profound lesions undergone by the cholera vibrios under the influence of the peritonaeal fluid of the immunized animal.

“On the other hand, one is compelled to the conclusion that the granular transformation is due, as we shall see later, to a fermentative action of the peritonaeal exudation.”13

Some authorities have considered the leucocytes to be an essential part of the blood, in which case their dissolution should be a dangerous loss to the person concerned. In my opinion, however, the leucocytes are nothing more than body waste or refuse in the process of elimination, and their dissolution immediately places a liquid toxic poison in the blood with no means of preventing it being absorbed, wherever the blood goes, into any and all tissues. Hence the possibility that the brain, the heart, or other organs not intended to handle these toxic poisons might absorb some of them.

Have you ever seen two leucocytes that were the same size or shape? They appear to vary widely in both characteristics – looking, in fact, more like crumbled cheese than living tissues.

Germs in serums may attack the heart valves

Other authorities have described other dangers in the use of serums, for instance Dr E. C. Rosenow, then of the Mayo Clinic, said over 25 years ago that certain varieties of germs in serums used in his experiments had ‘an affinity for the heart valves’!14

He describes experiments in which he found that the green-producing variety of germs in the serums attacked the valves of the heart, while a certain hemolyzing variety attacked the body joints, thus causing rheumatism!

In November 1925, the Chicago Health Department stated that:

“more children of the ages of 10 to 14 die of heart disease in Chicago than of all other children’s diseases put together!”

If Dr Rosenow’s statements are true, do you wonder that Chicago children are dropping dead on the street, with all the serumization that is practised in our schools? In the olden days, it was very rare for a child of 10 to 14 years of age to die of heart disease.

Dr Frederick Hoffman, Consulting Statistician of the Prudential Insurance Company of America, said:

“Heart diseases in all civilized countries are the leading cause of death and of a vast amount of physical impairment. As far as it is possible to judge, the relative frequency of heart disease in proportion to population has everywhere been increasing during the last two decades, although evidence to this effect is more or less conflicting.”15

While most diseases that kill mankind off have gone down at an almost wonderful pace since sanitation was first introduced to the world, this particular one is increasing, and for some reason the authorities profess not to understand.

I would like to ask the reader to refer to some of the charts accompanying this text, for instance Figure 4 (p.81).

Note that those immigrants from countries having compulsory vaccination die off at a rate three to four times higher than immigrants from countries not having compulsory vaccination.

There is no doubt that there are other causes to be considered, such as sanitation, living conditions, diet, and that the relative vitality of the different races may vary, so why should these death rates seem to divide simply on their vaccination status? And granting this, why does heart disease lead all other diseases in the difference between the high rates and the low?

It seems to me that this chart alone is very conclusive evidence that the statements we have quoted in this chapter, as to biologicals causing both tuberculosis and heart disease, are correct.

In regard to Italy, which passed a law requiring the compulsory vaccination of infants in 1888, we still class it in the ‘without’ column, because in 1910, the time of this census, probably not over 25% of the immigrants in New York State would be under 22 years of age and thus affected by the law, and it is very likely that the law was inefficiently enforced for the earlier years, thus allowing many to escape. Furthermore, all of those vaccinated would still be too young for the full effects of any injurious biologicals to become fully developed by 1910; hence Italy’s inclusion in the unvaccinated column.

Statistics of later years seem to indicate that Italy now has death rates comparable with other countries having compulsory vaccination, which can only serve to strengthen the idea that the fad for serums is the cause! See Figure 3 (p.80).

Dr Rosenow also speaks of yet other troubles that may follow the use of biologicals. In a series of articles dealing with the influenza epidemic of 1918 and published in The Journal of Infectious Diseases, and also in the Collected Papers of the Mayo Clinic, Vols 10, 11, and 12, he describes many changes in serums or in patients which rendered the serum useless.

In Vol. 10, p.919, he observes of the pneumococcus-streptococcus group, of which he thought mutation forms were responsible for the 1918 pandemic:

“… marked changes in morphology, growth characteristics, infective powers, and immunological reactions. Many of these changes appear to be true mutations.”

On page 949 of the same volume, he ascribed deaths following the use of certain serums to some change or mutation in either the serum or patient.

While, I believe, a serum is supposed to cure by ‘agglutinating’ all germs of that exact kind which it finds in the body, when there is a slight difference in germs, or changes occur, either in the patient’s germs or in those in the serum, no ‘agglutination’ takes place, and the patient is apt to die, unless sanitary or other measures are taken to save him.

Most regular physicians will say in such a condition that there is no hope, but if drugless physicians are called in, or if enemas are given, there is more than hope. In fact I believe two or three enemas a day and an exclusive fruit juice diet for a while would save the great majority of these cases. However, this is not meant to be a discussion of the treatment of disease, which is covered in other books.

That this change or mutation of germs is a very serious handicap in treating diseases by means of serums or vaccines is indicated all through the series of ten papers that Dr Rosenow published in Vol. 12 of the Mayo Clinic papers.

He says in Vol. 12, page 920, that the serum used on some guinea pigs ‘tended to localize in the lungs’.

In Vol. 12, page 1001, he says:

“Moreover, marked changes in the immunological condition as measured by agglutination tests have occurred in a number of strains following successive (intratracheal) animal passages.”

He added that when the changes occurred, ‘no good effects were noted’.

If passage through animal tissue will cause ‘marked changes in the immunological condition’, how can anyone know that passage through human tissues, for example from the arm into the body, will not do the same?

And where can you find a serum or vaccine that has not had an animal passage at some previous time? They are nearly all propagated in animals at present and a substantial percentage of all ‘passages’ seem to cause a change. In table 4 he shows 35 changes in 44 cases, and one of the other nine had changed in a previous experiment; that makes changes in over 81% of the tests!

This change is no accident; in fact, it occurs with great frequency, as Béchamp proved many years ago.

And these changes in the germs mentioned are of vital importance, as they often merely substitute a new disease for the one vaccinated against. Pasteur seemed to recognize the importance of this point as he vehemently denied its possibility to the very last, and made bitter personal attacks on Béchamp and other colleagues who opposed his ideas for this reason.

Now that this has been proven so overwhelmingly, we can see how a vaccine for any one disease could start some other disease through these mutated forms. We shall then need more serums for the new diseases, and so on, ad infinitum.

In the pamphlets Germ Mutation and Immunity, Artificial vs Natural, I give some important evidence indicating that the 1918 influenza epidemic was caused by mutation in vaccines used to ‘prevent’ typhoid in the armies in Europe.

When they inoculated against typhoid, they soon found that they had a para-typhoid on their hands, and the percentage of paratyphoid in those inoculated was identical to the second decimal place with the percentage of typhoid in those not inoculated.16 And when they gave two ‘shots’, one for each of these, they discovered a second paratyphoid, so to be scientific they called them ‘A’ and ‘B’.

And, as scientists must always be ‘scientific’, they then gave the boys three shots, one for each of the above diseases, whereupon they found a fourth ‘disease’ – influenza – and the world’s highest recorded death rate at that! The Surgeon General of the A.E.F. said of this ‘influenza’:

“The ordinary clinical picture of typhoid paratyphoid is frequently profoundly modified in vaccinated individuals … intestinal types of supposed influenza should always be considered as possible typhoid until proven otherwise. Vaccination is a partial protection only, and must be reinforced by sanitary measures.”17

Furthermore, supposing that there is no change and that a serum or vaccine ‘agglutinates’ perfectly, what proof have we that it will either prevent or cure any disease?

Elie Metchnikoff, says:18

“The most carefully studied case of the relations between natural immunity and agglutination is of that encountered in the anthrax bacillus. We owe it to Gengou,19 who at the Liege Bacteriological Institute carried out a very detailed investigation of this question.

“He showed that the bacillus of Pasteur’s first anthrax vaccine is agglutinated by the blood serum of a great number of animals. But he also showed that the serums which have the greatest agglutinative action on this bacillus do not come from the most refractory species. Human serum agglutinates most strongly the bacillus of the first vaccine (in the proportion of one part of serum to 500 parts of culture) but man is far from being exempt from anthrax.

“‘Pigeons’ serum, on the other hand, is completely without any agglutinative power, although this species resists not only the first vaccine but very often virulent anthrax. The serum of the ox, a species susceptible to anthrax, is more agglutinative (1:120) than that of the refractory dog (1:100).

“All these facts fully justify the conclusion formulated by Gengou that we cannot establish any relation between the agglutinating power and the refractory state of the animals to anthrax … this conclusion may be extended to the phenomena of the agglutination of micro-organisms and to those of natural immunity in general.”

It is quite likely that most physicians will acknowledge that when the changes in a germ as described above occur, there is practically no possibility of it preventing or curing any disease, and while these changes may not run as high as 80% with all biologicals, nevertheless we have shown that it can and does occur with sufficient frequency to render all such methods utterly unworthy of confidence, and unfit to rely on to any degree.

And Professor Metchnikoff’s statement that agglutination is of no value as an indication of immunity or curing power seems to wipe out any small remaining chance that serums can be beneficial, under any conditions.

In other words, it seems that when we get vaccinated and fail to catch any disease afterwards, it is either only an accident, or is due more to our natural immunity than to the serum.

1. On the Artificial Production of Tubercle in the Lower Animals, pub. McMillan, 1868

2. It is important to note that all these experiments were completed before the appearance of my last report.

3. Cohnheim and Frankel, Untersuchungen uber die ebertrayberkit der Tuberculose aut Thiere, Virch. Archiv. Bd. xlv, 1868, p.216

4. Fox loc. cit., p.20, Cohnheim l.c.p. 219

5. Microbe Hunters, p.131

6. ibid, p.193

7. Medical Journal of Australia, Sydney, Oct. 22, 1921

8. American Journal of Public Health, July, 1928

9. Journal of Agricultural Research, July 31, 1916, p.675

10. Immunity in Infectious Diseases, p.87

11. Munchen. med. Wchnscher, 1900, s.1193

12. Ann. de l’Inst Pasteur Paris, 1899, vol. 13, p.273; ibid, 1901, vol. 15, p.312

13. Immunity in Infectious Diseases, p.212

14. Journal of Infectious Diseases, 14, pp.1-32, 1914

15. The Problem of Heart Disease, The Spectator, Aug. 29, 1929

16. Journal of the A.M.A., p.267, July 28, 1917

17. Army Circular quoted in U.S.P.H. Report, March 28, 1919, pp. 611, 614, 619

18. Immunity in Infectious Diseases, p.203

19. Arch internat de Pharmocodyn, Gand et Paris, 1899, Vol.6, 299; Ann. de L’Inst. Pasteur, Paris 1899, Vol. 13, p.642

Chapter 8 ♦ Animal Serology: Anthrax

Ethel Hume says1 that a Frenchman named Delafond in 1838 announced that small rod-like objects were to be found in the blood of animals having splenic fever or charbon (now called anthrax), and when Pasteur brought out his one specific germ for each kind of fermentation, Devaine suggested that these little ‘rods’, which he named bacteridia, might be parasites and the cause of the splenic fever. However, his experiments were contradictory and it was not proven. Later, in 1878, Koch made some studies in which he discovered a formation of spores among his ‘bacteridia’.

When Pasteur heard of this, he declared:

“Anthrax is, therefore, the disease of the bacteridium, as trichinosis is the disease of the trichina, as itch is the disease of its special acarus.”2

He claimed that the blood of an animal vaccinated with anthrax serum contained no other organisms but the bacteridia. As he considered these exclusively aerobic, the blood must be imputressible, because putrescence, he believed, was due solely to an anaerobic germ. (Later, when the Professors of the Turin Commission drew contrary conclusions from similar experiments, he charged that they had used sheep whose blood was ‘septic’ as well as tainted with anthrax!)

He claimed that a mixture of aerobic germs (the bacteridia), and anaerobic germs (of putrefaction) would ‘neutralize the virulence’ of the bacillus anthracis and, if injected into animals, would protect them from infection.

In reality these two germs are only different developments or outgrowths of Béchamp’s microzymas, and should have much the same effect anywhere, namely that of scavengers of dead tissues or waste. Their action should be similar, and not counteractant to each other, as is indicated in Chapter 2.

Dr Colin, another member of the Academy, promptly challenged Pasteur’s statement on the grounds that anthrax was sometimes found in a virulent stage, yet devoid of the ‘bacteridia’.

In the next session (March 12, 1878) Dr Colin charged that Pasteur had suppressed two statements in the printed record that he had made on the floor during the prior session, i.e. that ‘the bacteridia of anthrax do not develop in the blood of healthy animals’ and that ‘the bacteridia will not supply germs to the organisms,’ which left Dr Colin’s criticism of these statements ‘in the air’, and, in addition, he charged that Pasteur had deliberately falsified the records of other criticisms Dr Colin had made.3

On April 30, 1878, Pasteur read before the Academy of Science a paper entitled The Theory of Germs and their Application to Medicine and Surgery4, which also bore the names of Messrs Joubert and Chamberlain as co-authors. This was his first attempt to sell the ‘germ theory’.

In this, among many false claims, was the statement that he had discovered ‘the fact that ferments are living beings’ – giving no credit to Béchamp whatever.

This paper also claimed that an infinitesimal quantity of their last produced culture was capable of producing anthrax with all its symptoms; yet their first experiments with it were failures; as the cultures, when sowed, produced a small spherical germ that was not even virulent, instead of the typical anthrax rods that were expected!

This was probably a true mutation but was not so recognized, the authors apparently believing it to be the result of an impurity getting into their cultures.

The London Times of August 8, 1881, about three years later, quotes Pasteur as saying before a sectional meeting of an international medical congress in session there:

“… in the study of micro-organisms there was an ever present source of error in the introduction of foreign germs, in spite of the precautions that might be taken against them. When the observer saw first one organism and afterwards a different one, he was prone to conclude that the first organism had undergone a change. Yet this might be a pure illusion … the transformation of a bacillus anthracis into a micrococcus did not exist.”5

Note that he said this 21 years after Miss Nightingale made her famous statement that any germ could turn into another, as previously quoted on page 3.

And when their own experiments failed to bear out their claims that their culture would produce anthrax or any of its symptoms, and the germs that were produced had no resemblance to the anthrax germ, either in appearance or virulence, why should others believe that they could prevent anthrax through the use of this ‘culture’?

But Paul de Kruif, in Microbe Hunters, a glorification of many famous pioneer serum faddists, paints a most astonishing picture of Pasteur’s work on anthrax, and gives many startling details regarding the facts of the matter.

After describing the silkworm failure, he says:

“But one of Pasteur’s most charming traits was his characteristic of a scientific Phoenix, who rose triumphantly from the ashes of his own mistakes … so it is not surprising to find him, with Reux and Chamberlain, in 1881 discovering a very pretty way of taming vicious anthrax microbes and turning them into a vaccine.”6

He describes Pasteur’s demonstration of his anthrax vaccine at Pouilly-le-Fort in May and June of that year in great detail, including the elaborate preparations, and he dwells on the fact that this experiment was framed by his enemies to destroy him, and that Pasteur realized that he was cornered, that he must succeed, or else abandon his work on germs.

It seems to me that we have now seen too many cases of deceitfulness, prevarication and deliberate fraud on Pasteur’s part to place much confidence in his good faith under such conditions, and in fact one is justified in looking with suspicion on this experiment. Here were 48 sheep – 24 supposed to be vaccinated, lived, while 24 not vaccinated, died. In such a number the treatment might be differentiated quite easily. He could easily have injected the unvaccinated sheep with a slow poison and he might have used pure sterile water, or a syringe with a perforated piston, in a pretended injection of the vaccinated sheep!

This ‘miracle’, as de Kruif describes it, seems to be the only success in a long series of failures; the single result that gives the only real support to Pasteur’s claims. After all the double-dealing and fraud that we have proven elsewhere, are we not entitled to be sceptical of this? Does not his past conduct suggest that he could have been loading the dice? And he does not seem to have been able to repeat the success elsewhere!

De Kruif says of this fact (p.165):

“Gradually, hardly a year after the miracle of Pouilly-le-Fort, it began to be evident that Pasteur, though a most original microbe hunter, was not an infallible god. Disturbing letters began to pile up on his desk; complaints from Montpotheir and a dozen towns of France, and from Packisch and Kapuvar in Hungary. Sheep were dying from anthrax – not natural anthrax they had picked up in dangerous fields, but anthrax they had got from those vaccines that were meant to save them! From other places came sinister stories of how the vaccines had failed to work – the vaccine had been paid for, whole flocks of sheep had been injected, the farmers had gone to bed breathing ‘Thank God for our great man Pasteur’, only to wake up in the morning to find their fields littered with the carcasses of dead sheep, and these sheep – which ought to have been immune – had died from the lurking anthrax spores that lay in their fields.*

“Pasteur began to hate opening his letters, he wanted to stop his ears against snickers that sounded from around corners, and then – the worst thing that could possibly happen – came a cold, terribly exact, scientific report from the laboratory of that nasty little German Koch in Berlin, and this report ripped the practicalness of the anthrax vaccine to tatters. Pasteur knew that Koch was the most accurate microbe hunter in the world!

“There is no doubt that Pasteur lost some sleep from this aftermath of his glorious discovery, but God rest him, he was a gallant man. It was not in him to admit, either to the public or to himself, that his sweeping claims were wrong…

“What a searcher this Pasteur was, and yet how little of that fine selfless candour of Socrates or Rabelais is to be found in him. But he is not in any way to be blamed for that, for while Socrates and Rabelais were only looking for truth, Pasteur’s work carried him more and more into the frantic business of saving lives, and in this matter, truth is not of the first importance.

“In 1882, while his desk was loaded with reports of disasters, Pasteur went to Geneva, and there before the cream of disease-fighters of the world, he gave a thrilling speech, with the subject: How to guard living creatures from virulent maladies by injecting them with weakened microbes.”

And according to de Kruif, Koch made a devastating attack upon Pasteur’s statements in a paper published shortly after this, in which he charged that practically all of Pasteur’s claims for his anthrax vaccine were false, that his vaccines were not pure, and that he had concealed the bad results that had followed the wholesale use of the vaccines. He closed with:

“Such goings-on are perhaps suitable for the advertising of a business house, but science should reject them vigorously.” (p.168)

De Kruif adds:

“Then Pasteur went through the roof and answered Koch’s cool facts in an amazing paper with arguments that would not have fooled the jury of a country debating society.”

How can de Kruif so praise a man, and describe the ‘miracle of Pouilly-le-Fort’ as ‘amazing as any of the marvels wrought by the Man of Galilee’, after giving such devastating evidence that his work was a failure, his ideas false, and the man himself deliberately dishonest, making false claims and concealing the extent of his failures?

* Or was it from the vaccines? – R. Pearson.

In 1881, the Sanitary Commission of the Hungarian Government said of the vaccine viruses used in the anti-anthrax inoculation:

“The worst diseases, pneumonia, catarrhal fever, etc., have exclusively struck down the animals subjected to injection. It follows from this that the Pasteur inoculation tends to accelerate the action of certain latent diseases and to hasten the mortal issue of other grave affections.”7

Plainly it failed in their tests also, and the Hungarian Government forbade its use in that country.

It was not long before his vaccine was proven a failure elsewhere as well. In March 1882, a commission composed of members of the faculty of the University of Turin, Italy, conducted tests regarding the value of this anthrax prophylactic. A sheep having died of anthrax, after they had vaccinated some other sheep with Pasteur’s cultures, they inoculated both these vaccinated sheep and also some unvaccinated sheep with the blood of the dead sheep. All of the sheep, both vaccinated and unvaccinated, subsequently died, proving the vaccine utterly worthless.

After about a year of dispute and passing the buck by correspondence, the Turin professors published a pamphlet in June 1883, containing some of Pasteur’s contradictory statements together with their cutting criticisms thereof, under the title Of the Scientific Dogmatism of the Illustrious Professor Pasteur, which was signed by six professors of high standing. This, by citing contradictory statements Pasteur had made in different papers, along with their comments, just about destroyed his theories on anthrax.

This paper was translated into French, but Pasteur, with some adroit dissimulation, managed to survive the blow, and went on pushing his anthrax vaccine.

He soon had bacteriological institutes for experiments and the production and sale of his various serums and vaccines established in many parts of the world, the one in Paris being probably the first.

In 1888 an institute in Odessa, Russia, sent some anti-anthrax vaccines to Kachowka in southern Russia, where 4,564 sheep were soon vaccinated, and 3,696 of them promptly turned up their toes and died; a death rate of 81 percent, and from a supposed ‘preventative’ vaccine at that!

Dr Lutaud says in Etudes sur la Rage (p.419) that Pasteur was compelled to compensate many owners in France for animals killed by his vaccines, but his work went on.

Foot and Mouth Disease

Mr C. M. Higgins, of drawing ink fame, of Brooklyn, N.Y., some years ago wrote a book entitled Horrors of Vaccination in which he drew attention to the fact that official publications of the United States Government ascribed several epidemics of foot and mouth disease in this country directly to the use of vaccines or serums; especially those of 1902, 1908, and 1915.

The Chief of the Bureau of Animal Industry of the US. Department of Agriculture says in his report for 1902:

“Most veterinary text books state that foot and mouth disease is a mild infection and that only 1 or 2 percent of the animals attacked die from it, the reader being left to infer that the losses do not exceed 2 or 3 percent of the value of the animals. Such a conclusion would be a grave mistake.”

However, it seems to have been mild before its cause was traced to vaccines. The Secretary of Agriculture says in the department 1914 Year Book, page 20:

“There were outbreaks of foot and mouth disease in this country in 1870, 1880, 1884, 1902, and 1908. Since the close of the fiscal year 1914, the sixth outbreak has occurred. The first three, those of 1870, 1880 and 1884 were comparatively trifling. Those in 1902 and 1908 were more grave. The present one is the most serious and extensive of all.

“In 1902, the outbreak occurred in the New England States. In 1908 it originated in Detroit. The origin of each of these new outbreaks was traced to the importation of vaccine virus for the propagation of vaccine for use in vaccinating people against smallpox. The vaccine was imported from Japan where the foot and mouth disease exists. Each of these outbreaks was stamped out by methods which have proved most effective in preventing the disease from gaining a footing. These methods involved the killing of all infected and exposed animals, the burying of the carcasses, and the thorough disinfection of all premises with which the animals may have come in contact.”

The first part of the 1914 outbreak was ascribed to ‘an imported article used in tanning’ (hides?) but when this was stamped out, a recurrence occurred near Chicago, in August 1915, that was traced to a Chicago laboratory making hog-cholera vaccines. Foot and mouth disease was found in 8 of 11 herds that had used this vaccine.

The Secretary of Agriculture says of this in the 1915 Year Book (p.27):

“It seems certain that this infection was produced by contaminated hog-cholera serum prepared in Chicago, in October 1914, at an establishment where the disease had not been known to exist at any time.

“… pending investigation, all shipments of serum from Chicago were prohibited. It was found that some of the product of the establishment had been used on 11 herds of hogs.

“… infected hogs were found in eight of the herds and all 11 herds were slaughtered at once.”

Although they had found the disease in 8 herds on which the vaccine had been used, they decided to ‘test’ the serum, and what a test!

They knew, or were very sure, that the vaccine had given the hogs the foot and mouth disease, yet the first four tests on a total of 52 animals were all negative, but they had plenty of perseverance, and in the fifth ‘test’ and on the 62nd animal tested, they found foot and mouth disease!

If it took ‘tests’ on 62 animals to obtain proof that a vaccine that had already caused the disease could do so again, how can anyone know that it would not take two or three or more times 62 ‘tests’ any other time, assuming, of course, that these are tests, which, again, I don’t believe!

And after such a failure, how can any doctor or veterinarian consider any tests, such as the Schick, Dick, Tuberculin, Wasserman, etc., to be of any value whatsoever?

With all the evidence we have given that germs can change their characteristics, from Miss Nightingale and Professor Béchamp, to Lohnis, Rosenow and others, how can anyone expect a germ to remain constant through any ‘test’ or remain true to its original characteristics after being ‘tested’?

The Secretary of Agriculture says of these so-called ‘tests’ – on the same page:

“This is regarded as proof that the suspected serum actually was infected. Why the standard test used on 61 of the animals failed to reveal this fact is a matter for scientific investigation, and the bacteriologists of the department are at work on the problem. At the time of manufacture 0.5% of carbolic acid was mixed with the serum as a preservative. It is now believed that the acid, acting as a germicide, may have attenuated or partially destroyed the virus, so that tests previously considered safe failed to establish the presence of the infection.”

If they had no better luck than Pasteur had with his anthrax tests, it will be a long time before they find out very much!

As the average serum is only some toxic decomposing proteins, and some germs that are really reworkers of dead tissues or waste, but which the doctors believe to be the cause of the dead tissues they are found with, the germs are very apt to change their characteristics as the toxins break up, just as they have repeatedly been shown to do elsewhere in nature.

Consequently, many serums would not remain constant through 61 tests, nor would anyone who sells serums to the public be likely to make 62 tests before telling their customers that it was pure serum!

Even after it is ‘tested’ it may change in storage, and how do they know when they have the right germ in the serum anyway, as the best authorities admit that some germs, such as the smallpox germ, have not been isolated?

The Secretary of Agriculture says (of hoof and mouth disease) on page 29 of the same volume:

“Up to the present time, the germ has not been identified, although the scientists of Europe have studied the disease exhaustively for years.”

They killed 168,158 animals valued at about $5,676,000 to suppress the 1914-15 epidemic.

Circular No. 325 of the Agricultural Department says:

“Immunization in the 1914 outbreak was out of the question, as the only serum thus far produced gives but a passing immunity of only a few weeks duration, unstable at best.”

Mr Higgins pointed out that the disease is more prevalent in countries that have compulsory vaccination than in others.

The U.S. Dept. of Agriculture quotes Dr Loeffler, head of the department handling the trouble in Germany, as saying before the 7th International Congress of Veterinary Surgeons at Baden Baden in 1899:

“Foot and mouth disease is spreading more and more every year, and every year it costs the German Empire enormous sums. Necessary measures have been taken with the greatest care; suspected grounds have been closely quarantined; this measure had been extended to whole communities and even to entire districts; disinfection had been carefully carried out; and notwithstanding all this, the disease kept spreading.”8

Foot and Mouth Disease in Germany

8 1

The Foot and Mouth Disease Commission of the U.S. Department of Agriculture published a chart9 showing the trend of foot and mouth disease in Germany from 1886 to 1924, which is reproduced at right.

Note the tremendous increase in deaths that accompanied the first general use of serums in 1920.

The U.S. Department’s Farmers’ Bulletin No. 666 says:

“Foot and mouth disease has prevailed in Europe for a great many years and has occasioned tremendous economic losses there.

“In Italy, France, Switzerland, Germany and Russia the plague has existed so long and has gained such a foothold that it is economically impossible to fight it with the American methods of slaughter and disinfection.”

In Germany in 1911, 3,366,369 cattle, 1,602,927 sheep, 2,555,371 hogs and 53,674 goats were affected, or 7,578,371 animals of a total number of about 51,319,000 farm animals in the country at that time. As the chart indicates that about 247,000 farms were affected that year, this would give about 30.6 animals per farm. If the 1920 figures of 746,571 farms affected averaged the same, it would run to nearly 23,000,000 animals, close to half the number of animals in Germany! They used serums this year also, which probably helped spread it.

The same bulletin quotes one scientist as saying:

“… unless all the affected farms were absolutely isolated and the movement not only of livestock but of persons absolutely prohibited, the disease could not be stamped out. Such a quarantine is of course utterly impossible to enforce.”

Italy, France, Germany and Switzerland have compulsory vaccination, hence large vaccine plants that can spread the disease, as occurred in the cases cited in the United States.

And of course neighbouring states with or without compulsory vaccination would be overrun by importation from these countries, though some, such as England, kept it out pretty well.

Other places where vaccination is pushed, such as Brazil, also have the disease, while Canada, the United States, Mexico, Australia and New Zealand, all of which are comparatively free from intensive vaccination drives, also seem to have only sporadic attacks of foot and mouth disease, which are generally easily stamped out.

How can the ‘scientists’ account for this?

Rabies or Hydrophobia

According to Farmers’ Bulletin No. 449 of the U.S. Agricultural Department, no one can catch rabies from an animal that bites them unless the animal has the disease. Furthermore, less than 15% of those bitten by a rabid dog and not treated will generally contract the disease. This is very different from the hullaballoo generally raised by the self-styled ‘regular’ doctors, and especially by health officers, over every dog bite they hear of. In an official publication such as the Farmers’ Bulletin, this is quite an admission; unofficial and anti-vivisection sources of information generally place the percentage much closer to zero.

Bulletin No. 65 of the U.S. Hygienic Laboratory at Washington also admits that those who die after treatment die earlier than untreated cases! It says:

“Nitsch has pointed out that in a large series of cases the deaths in spite of the Pasteur treatment occurred on average earlier than in untreated persons (64.5 to 90 days).

“There is some reason to believe that the rabies virus as it occurs in nature varies much in virulence, and that this is in some way related to the geographic distribution.” (p.21)

To anyone who read Chapter 7 it will be evident that (assuming it has value), one should not use a serum from a distant location if this is true, as the possibility of ‘agglutination’ would be very small where there were such variations. And to this they add:

“Inoculation with spinal fluid obtained during life is wholly unreliable as it usually fails even in true cases of rabies.” (p.36)

The New York Anti-Vivisection Society has published several pamphlets from which the following information is taken. They state that rabies is a very rare disease except where dogs have been injected with rabies serum, in which case it very often develops.

According to their views, a dog unable to find green grass to eat in winter is very apt to develop worms or maggots, or both, in the intestines, often perforating them, and driving the dog frantic. In this condition the dog will bite at everything blindly, foam at the mouth, and run amuck generally, refusing water and seeking solitude. Hay, grass, hide or bones fed to the dogs will cause the irritable conditions to disappear.

There are no real grounds for supposing that madness, as found in humans, occurs in dogs, nor can it be proved that the bite from a distracted animal can produce madness in anyone bitten. Further, so-called rabies can be shown to be the direct result of serum injections.

Competent authorities claim that in so-called ‘real’ rabies, a dog never foams at the mouth, but has a small amount of brownish stringy discharge hanging from the lips, and the eyes have a fiery glare.

In epilepsy, the dog trembles, his jaws champ violently and his voluntary muscles are powerfully convulsed; there is a copious discharge of white frothy saliva; he utters sharp cries and when recovering from the fit, the eyes are dull and stupid. This might be due to fright, or heat in summer.

They quote doctors of unquestionable authority as saying that no rabic germ has been found; and that finding so-called Negri bodies is no proof that the dog has rabies; as

“they are found when all symptoms are absent and when all are present, so the diagnosis of rabies is pure guesswork,”

according to J.A. McLaughlin.

Even by A.M.A. standards, no successful serum can be made without the right germ, so this might account for the large number of deaths that follow the Pasteur treatment.

Some doctors say the bite of a rabid dog is absolutely harmless to man. C. W. Dulles, M.D., a famous authority on dog diseases and hydrophobia who looked up the records in many cities, says over a million dogs and cats were handled by dog catchers in 14 years, with many thousands of bites, but no treatment – and not a single case of hydrophobia appeared in these cases.

He and other doctors had posted for years standing offers of $100 to $1,000 for a genuine case of dog hydrophobia and had no claimants, though thousands of dogs were being killed yearly because of scares; one place claiming that 92% of those killed in one year had hydrophobia!

These doctors say chaining or muzzling a dog that has always been free is apt to cause the very irritability we want to avoid.

Pasteur’s treatment causes rabies

In man, they say the death rate in France in cases of so-called rabies is 19 per 100 – the highest in the civilized world, and the same as before the Pasteur Institute was established; and cases of hydrophobia have enormously increased, while just across the Rhine in Germany, hydrophobia is almost unknown.

The year before Pasteur started his treatments, there were four deaths from hydrophobia in Paris; the year after there were 22! Not only France as a whole, but each department of France, and in fact every country that has allowed the Pasteur ‘treatment’ to be introduced, have all shown a sharp increase in the number of deaths from hydrophobia after such introduction!

In England there were several Pasteur Institutes doing a thriving business prior to 1902, when a commission was appointed to investigate rabies and the serum treatment, and the Institutes were abolished. They have had no hydrophobia since.

They claim that over 3,000 people died in England before 1902 after being bitten by dogs and then taking the Pasteur treatment, while more recently the London Hospital treated 2,668 persons bitten by dogs without using the Pasteur treatment, and none of them developed hydrophobia!

While these are not complete figures for England, there are nearly 6,000 cases of dog bite treated in institutions; and of these only those who had taken the Pasteur treatment died. Why not try something different?

And there has never been a case of hydrophobia in Norway, Sweden, Iceland, Denmark, Holland, Belgium, New Zealand and Australia, because those countries will not tolerate a Pasteur Institute within their borders.

They say the Pasteur treatment is very often the cause of rabies, is always dangerous, sometimes even murderous, and is never beneficial.

J. W. Dodson, M.D., of Brockport, N.Y., wrote years ago:

“If people would only think for themselves and not blindly follow the agitator or grafter, we would soon be relieved of this pest, rabies.”

For a safe, sane and logical treatment that has saved patients with rabies for over 100 years, we would recommend the Buisson Bath, a hot vapor or steam bath that is fully described in Drugless Cures by this author.

The tuberculin test

As the so-called tuberculin test has been rather fully discussed in the pamphlet The Tuberculin Test a Fraud, and in Chapter 7 of this volume, it seems hardly necessary to say more on this subject here.

Needless to say, it is as big a fraud as a ‘test’ on animals as it was as a ‘cure’ for humans, and there is a great deal of substantial evidence that the testing vaccine (or its needle) causes tuberculosis in cows and other animals, as it did in the human subjects used in Koch’s experiments.

It should be absolutely forbidden, and those who use it should be barred from practice.

1. Béchamp or Pasteur?

2. The Life of Pasteur, Rene Vallery Radot, p.260

3. Béchamp or Pasteur?, p.198 (original edition)

4. Comptes Rendus, 86, p.1037

5. The Life of Pasteur, by Rene Vallery Radot, p.329

6. Microbe Hunters, by Dr Paul de Kruif, p.157

7. Béchamp or Pasteur?, Ethel Douglas Hume, p.346 of this volume.

8. 1920 Year Book, U.S. Dept of Agriculture, p.651

9. Report of the Foot and Mouth Disease Commission, Dept of Agric. Bul. No 76, p.18

Chapter 9 ♦ Statistics

In any discussion of the value of a remedy or preventative for any disease, actual statistics of the results that have followed the use of such remedy or preventative in the past should be of great value in judging it, especially when the trend over a long period of years can be charted graphically.

Hence it seems proper to consider what a chart showing the death rates both before and after the introduction of some of these biological treatments, might indicate; especially when the results can be compared with the general trend following other methods of treatment of more or less similar diseases.

For this reason, this chapter contains several charts showing the death rates of several diseases both before and after the use of biologicals, as well as some of the death-rates of similar diseases with and without the use of biologicals.

These give an astounding confirmation of our contention that all biologicals are not only utterly useless, but are actually very harmful, and we urge a careful study of the charts.

These charts, together with the information that follows, should convince even the most faithful followers of Pasteur that the so-called ‘germ theory’ and the use of any and all biologicals is, as Dr Hudson says, wrong, and should be completely abandoned immediately.

FIGURE 1: Smallpox And Vaccination

In Leicester, England

9 1

Smallpox and vaccination in Leicester, England

Note how every increase in vaccination as indicated in the lines in the right hand column was followed by an increase in the smallpox death rate.

FIGURE 1: Smallpox And Vaccination In Leicester, England

Note that in this chart during all the years that vaccination was actively pushed, through compulsory laws, etc., smallpox returned again and again, and usually after an increase in vaccination; culminating in the great epidemic of 1872. This came after a most intensive four year effort to completely vaccinate the population under a law (of 1867) that made refusal to submit to it a penal offense. And it came just after the supreme Medical Officer of England had announced that 97.5% of the population had either had smallpox or been vaccinated.

The worst epidemic came just when they had obtained the utmost in ‘protection’! Also note that since this time, while vaccination has dropped off in Leicester, smallpox deaths have also dropped off, and in this case to none!

While the public has so lost confidence in vaccination that less than 6% of all newborn children in Leicester for the past 20 years were vaccinated, there has not been one single death from smallpox in the last 33 years of the records, from 1905 to 1938 inclusive.

There is probably no city or country having compulsory vaccination in the world that can show a record like this.

This chart indicates that vaccination is not only not a preventative, but that is probably an active and important instigator of smallpox.

We might mention here that Dr R. Garrow, Medical Officer of Health for Chesterfield, England, asks why it is that the case mortality rate from smallpox in all persons over the age of 15 in England and Wales for the years 1923-6 was five times as high in the vaccinated (0.3%), as in those who were unvaccinated (0.06%)!1 (He used official figures, and calculated only to one and two decimals; when figured to four decimals the rates become 0.324% and .0578%, nearer six times, the ratio between them being 5.6055 to 1. Other years seem to lean the same way, but to a lower ratio.)

If serums and vaccines have any value, as the pus-doctors have promised for so many years, why is not the ratio the other way around?

The Vaccination Inquirer of London, England,2 says that in Brazil, where they have rigid compulsory vaccination laws, and most energetic compulsion, the death rate from smallpox per 100,000 population in Rio de Janeiro for 1913–1922, a 10 year period, was over 600 times as high as that of London, where opposition is strong and the exemption laws are widely used!

If these are only ‘accidents’ as the doctors undoubtedly will claim, they at least prove that vaccines are useless; but my contention is that the use of animal pus injections is the cause of the higher figures.

FIGURE 2: Smallpox And Vaccination In Japan

9 2

Death rates from various diseases in Japan.

Note how the increases in vaccinations in 1897 and 1908 were followed by increases in tuberculosis, heart disease, pneumonia, etc.

FIGURE 2: Smallpox And Vaccination In Japan

As Japan has had the most intensive and thorough vaccination practice of any country, probably for the last 60 or 80 years, any bad effects that might be due to serum or other biologicals ought to be registered here more fully and plainly than in less serumized countries.

To get an idea as to what the effects of biologicals have been here, I have charted the death rates of several diseases in Japan for the period since 1885, from figures in the Annual Reports of the Statistical Bureau, filling in one or two gaps from the Japan Year Book, and have also charted the total number of vaccinations in each year.

Vaccination was introduced into Japan in 1849, and it is said that ‘the people eagerly adopted it’. The first vaccination law was passed in 1874 and compulsory laws were passed in 1876, 1885, and 1909, each being more stringent than the previous one.

Now, I understand, a baby must be vaccinated within 90 days of birth, and again during the second and tenth years. As there have been over 187,679,000 vaccinations in Japan between 1885 and 1928 inclusive, any injurious effects of vaccination should be fully apparent here.

In 1890 animal lymph was introduced, at which time the death rate from tuberculosis and other lung troubles began to climb, and increased almost continuously for 28 years. Note that the peak years of vaccination are all followed by a rising death rate in these troubles, and that these tend to drop off when the number of vaccinations has been reduced for some years.

One recent yearbook stated that the race was ‘degenerating’ because the mothers had become negligent(!) but I believe the use of serums is more likely to be the cause of any ‘degeneration’, as well as the present high death rates from tuberculosis, etc.

Note also that in recent years, with no smallpox scares and no extraordinary vaccinations, the death rates of all diseases charted have turned definitely downward as the total number of yearly vaccinations has come down. This lends additional support to our argument.

The editor of the Year Book died in the early 1930s, and later figures are not uniformly compiled, nor all available, hence the chart – made in 1931 – has not been brought up to date.

FIGURE 3: Vaccines and Tuberculosis

at School Ages

9 3

Vaccines and Tuberculosis at school ages

Note how much higher the death rate is in countries having compulsory vaccination, such as Japan and Italy, than in countries without compulsion, such as England, Wales and New Zealand.

FIGURE 3: Vaccines and Tuberculosis at School Ages

This chart bears out the view that biologicals can do the young no good. Note that the countries without any compulsory use of biologicals have the lowest death rates from tuberculosis among the young, while Japan stands at the opposite end, combining the most intensive compulsion known with the highest death rate.

FIGURE 4: Vaccines and other diseases

9 4

Mortality from highly fatal diseases among various groups of immigrants in New York State in 1910

Note that much higher death rates prevail among migrants from countries having compulsory vaccination (on left side of the diagram) than among immigrants from countries without compulsion (on the right side).

FIGURE 4: Vaccines and other diseases

In the first three charts we showed that biologicals not only did not prevent smallpox, but on the contrary actually seemed to increase it, and furthermore, also increased tuberculosis in a substantial manner.

In Figure 4 we show that any kind of biological given for any one disease can have a most astonishing effect in increasing other diseases with which – if we believe the pus-doctors’ theories – it should have absolutely no connection.

The reader should read Chapter 7 and study Figures 2, 3 and 4 together to appreciate how true are the statements made in that chapter.

The statistics in Figure 4 are for all the foreign-born immigrants from the countries noted who were living in New York State in 1910. There were from 340,000 to 560,000 persons included in each national group, hence they should give a very reliable picture of the resistance to disease of the various groups.

Italy, as previously noted, had a compulsory vaccination law passed in 1888, but is included in the unvaccinated column because only a small percentage of these immigrants were subject to the law because of their age, and all of those were too young and too recently vaccinated for the bad effects to be fully developed by 1910, as explained on that page. Note, however, that all death rates in the Italian group average above those of the other two countries in the same column, with the single exception of Bright’s disease.

Note also that in tuberculosis, all groups except Italy have a much higher death rate among the men, large numbers of whom are probably vaccinated with considerable frequency in the armies, than among women, very few of whom (except nurses), were likely to be vaccinated so often. Is there a plausible explanation for all the differences that are shown in these figures, apart from vaccination?

FIGURE 5: Death rates from important diseases

in the registration area of the United States

9 5

Death rates from important diseases in the United States

Note how the death rates from cancer and heart disease, which are aggravated by biologicals, have increased while others have gone down.

FIGURE 5: Death rates from important diseases in the registration area of the United States

As we saw in Figure 4 that serums seemed to increase other diseases, this chart was drawn to find the general trend in the United States.

Note the increase in heart disease and compare this with what we say in Chapter 7 (pp. 39–40) and with Figures 4 and 6. Cancer and nephritis seem to have a similar trend, though less marked.

Diptheria in Birmingham, 1890-1901

9 6

From the report of the Health Officer for Birmingham, 1901.

FIGURE 6: Diptheria, Scarlet Fever and Croup

in England and Wales

9 7

Diphtheria, Scarlet Fever and Croup in England and Wales

Note how the diphtheria death rate was held up well above the 1871-80 rate ever since antitoxin was introduced in 1893, while the death rates from scarlet fever and croup have consistently gone down at a rapid rate without the use of any biological.

FIGURE 6: Diptheria, Scarlet Fever and Croup in England and Wales

Note that while scarlet fever and croup have had an enormous drop in death rate since 1871 without any serums being employed, diphtheria had a large increase. In fact, there was a real epidemic running from 1893 for about seven years, the period when anti-toxin was pushed the hardest! And the death rate for 1921–29 still averages above that of fifty years earlier!

In other words, with the ‘help’ of the anti-toxin to fight diphtheria, there is an increase in the death rate over 1871–89, while scarlet fever and croup have both had astonishing drops in mortality, over 96% and 99.8% respectively, without any anti-toxins to help.

There are official statements that anti-toxin was introduced into England in 1894, but it hardly seems possible that such a poisonous biological as this would be introduced and made compulsory on a national scale without any prior knowledge or experience with it whatever. For this reason, I believe that it was tried out experimentally in parts of England at least in 1893, or even earlier, and in this way the epidemic that started that year could have been started and kept going until the exemption laws reduced the number of customers.

The report of the Health Officer for Birmingham, England, for 1901 contains the figure at the top of this page (Diptheria in Birmingham, 1890-1901).

There is no statement made as to when anti-toxin was introduced, but the rise in the death rate in 1895 would make me believe that it was in that year, and the Health Officer probably thought that this sudden increase at the time of the introduction of anti-toxin was merely a coincidence that was ‘just too bad’, and it might be best not to call attention to it.

Clearly, he believed in anti-toxin; when the drive to vaccinate every one passed its peak in 1897, he very quickly notes that the case mortality rate in diphtheria averaged 25% from January to June (when school let out) and only 20% from July to December. This 20% drop he ascribes to the fact that ‘the gratuitous distribution of anti-toxin was commenced in June, 1897’. He does concede that the school children were on vacation, with school vaccination programs suspended, might have had anything to do with it.

FIGURE 7: Diptheria and Anti-toxin

in Leicester, England

9 8

Diptheria in Leicester, England – Cases and Deaths.

Note how sharply the death rate rose after antitoxin was introduced in 1895, from an average of 62 per year for the previous 57 years to a high of 1514 deaths in 1900.

FIGURE 7: Diptheria and Anti-toxin in Leicester, England

While the diphtheria epidemic started in England in 1893, it did not reach Birmingham or Leicester until 1895, and the records show that anti-toxin was introduced into Leicester in 1895.

As the five-year periods did not bring out the great increase in diphtheria that followed the introduction of anti-toxin, and the corresponding reduction that occurred when the Exemption Laws were invoked for protection, I have charted these ten years individually. The group figures are in all cases yearly averages, hence comparable with the epidemic figures.

Note that here also diphtheria still has a higher death rate than that which prevailed for the 60 years before anti-toxin was introduced. Also, note how the curve of this epidemic follows the activities of the pus-squirters. Its rise and fall probably indicates fairly well the true effect of anti-toxin in many places where the figures are not published by the ‘physicians’ who know them. And it is still going on.

In 1935 Dr C. Millard, the Medical Officer of Health for Leicester, made a report on Inoculation Against Diphtheria to the Health Committee of the City Council3 in which he advised against ‘any action … encouraging inoculation of the general public’.

He notes rises in the death rate in Birmingham and other towns after a large number of inoculations, and expresses the belief that ‘much diphtheria is spread by carriers’ and that inoculation is ‘definitely increasing the numbers of carriers’.

He also quoted from a report4 of the Medical Research Council to the effect that inoculation seems to increase the death rate in many cases.

This report indicates that statistics gave no evidence of improvement from inoculation in large scale diphtheria rates for total populations up to 1929. It says:

“In France, the most inoculated country in Europe, the incidence of diphtheria continued to rise steadily from 1924 to 1930 … Craster (1931) relates that in 1921 an anti-diphtheria campaign was started in Newark, but was confined to the schools. At first, the result appeared promising, but in 1926 morbidity and fatality started to rise, and in 1929 Newark reported the highest diphtheria mortality in the United States for that year. It was noticed that the brunt of the disease was borne by the uninoculated children. Craster remarks:

‘I am confident that this occurred as the result of a general carrier condition among the immunized group in the home.’”(p.105)

This ‘confidence’ that pre-school age children died at such a high rate while the inoculated school children go entirely free sounds fishy to me. I think it is a case of misplaced confidence. It seems impossible for some doctors to doubt the germ theory.

Dr Millard mentions a reference to an increase in case fatality in Kansas from 4.9% in 1920 to 7.3% in 1929 and says

“Kinnaman (the State epidemiologist) believes that active immunization may actually increase the virulence of diphtheria bacilli”

and quotes the report as follows:

“Immunized carriers pass organisms of the more virulent type to non-immunized children, with the result that the fatality rate is increasing each year among non-immunized children who contract diphtheria.”

He also quotes the report to the effect that Detroit started a campaign in 1921, inoculating 3,000 a year for four years. In 1925 the yearly number was increased to 18,000 and in 1926 to 100,000. In this year there was:

“… a sudden rise in the attack and death rates of diphtheria. During the five years ending with 1930, the mean diphtheria death rate in much-inoculated Detroit was higher than that recorded in any other city in the United States.”

Dr Millard noted also that mortality from diphtheria for 1929 was 16.0 per 100,000 population, nearly four times the average of 4.1 for the last five years (1930–34) in Leicester, with no inoculations.

This sounds very much like what Dr J. W. Browne called ‘stirring up the embers and kindling the fire’ of tuberculosis.

Can you account for these increases with any other reasoning?

Dr W. Kellogg, director of the Bureau of Communicable Diseases, California State Board of Health, says the Schick Test should be abandoned completely. He adds:

“The percentage of errors in reading reactions in those who are protein-sensitive is frequently as high as 50%, even in the hands of the most experienced.”

What use is a test that is 50% wrong?

Dr J. Kilpatrick said before the Chicago Homeopathic Medical Society, December 4, 1928:

“I have never seen a person with a clean tongue and pure breath, who would take diptheria on exposure to it.”

FIGURE 8: Diptheria and Anti-toxin

in certain U.S. Cities

9 9

Deaths and use of toxin/anti-toxin in city schools in the U.S.

Note that the death rate declined until 1925, followed by a sharp rise in 1926/1927 in all cases, after the drive to sell anti-toxin started!

FIGURE 8: Diptheria and Anti-toxin in certain U.S. Cities

On January 1, 1926, the A.M.A-ites started a drive to abolish diphtheria by 1930 by injecting Toxin Anti-toxin (T.A.T.) into all school children in the country, and, of course, they started on the large cities where they had large health departments and many school doctors and nurses to push the sale of the pus. The figures when charted do not indicate that diphtheria has been abolished, nor does it seem likely to be! On the contrary, most of the larger cities had an increase for the years following 1925 that probably will average more that 100% above the 1925 figures.

This chart only includes a few cities in which the drive was particularly noticeable, and in which, if T.A.T. had any beneficial effects, a marked reduction in diphtheria should have occurred.

Yet 1930 is past, and diphtheria, instead of being abolished, is worst where T.A.T. was pushed the hardest!

The figures for 1915 to 1924 are taken from the 1924 volume of Mortality Statistics of the United States Census Bureau, while those for 1925 to 1928 are based upon the total deaths in the later volumes. The populations were estimated from the 1920 and 1930 figures by subtracting one-tenth of their difference from the latter figures, for each year prior to 1930. And as this would not be strictly accurate, no attempt was made to adjust the figures to July 1st populations, as is usual in figuring death rates. The 1923 figure for Detroit is estimated in the same way as it is omitted from the table.

The figures for 1929 and 1930 are deduced from a table in the 1925–30 report of the Chicago Health Department in the same way. These years are therefore not exact but should still be reasonably close to the true figures. The Youngstown figures for 1929 and 1930 were furnished by Mr J. Flood of Pittsburgh, Pa.

Note that this chart shows a very substantial trend downwards from 1915 to 1925 or 1926, when this big drive started, but following these two years there was a sharp increase which covered the years 1927–29 when the T.A.T. was most widely used.

I don’t know whether the drop in 1930 was due to a let-up in the drive, or to hard times reducing the amount of food eaten by the public, which might improve the average health as it did in Holland during the war, or to a drop in the use of serums. However, I do not believe this drop was due to anti-toxin, nor do I believe that anti-toxin or any other biological will ever be of any assistance whatever.

This chart was made in 1931, and in trying to add to it recently I failed to find later figures for these cities; possibly the allopaths would like to keep them out of circulation.

A Real Cure

In 1879, before anti-toxin was discovered, Dr J. Kellogg of Battle Creek wrote a book on diphtheria in which he deplored death rates of 40% to 75%, which he said had occurred in many places at that time, and recommended an eliminative treatment with which he claimed to have had no deaths in treating over 400 cases.

As the case mortality rates in Chicago and many other cities have been between 8% and 10% in recent years, it seems to me that Dr Kellogg’s methods would be a great improvement over any biologicals.

The evidence offered here should satisfy anyone that the use of animal pus to heal the sick is one of the greatest debacles in the history of mankind.

1. British Medical Journal, Jan. 14, 1928

2. Vaccination Inquirer, Jan. 2, 1928

3. The Medical Officer, 2nd and 9th March, 1935.

4. Special Report Series No. 195, Active Immunization against Diphtheria, S. Dudley et al.

Chapter 10 ♦ Real Immunity